So if children are mostly not affected by covid-19,(I know some children and young people get it and
are affected but with regards to numbers the vast majority are older folks)
My question would be what antiviral vaccine may have protected them.
Seems from what I've read the older you are the higher the risk the younger the lower the risk. See the widening window between vaccination and high risk?
Maybe the further we are from those childhood vaccines in our age the further from our immunity to this virus.
Posted by Lymetoo (Member # 743) on :
I don't think it has anything to do with their vaccines.
One is that children are more resilient and their lungs are in better shape. The real problem is how this COVID attacks the lungs.
Posted by Phoiph (Member # 41238) on :
Another theory is that children have higher levels of melatonin, which declines with age.
Posted by Garz (Member # 52095) on :
many things vary with age. probably 200 or so biomarkers or more are known to do so, so it could be any one of them.
But i think there is evidence that the variation of mortality of previous respiratory virus epidemics have been driven by the condition of the host's immune system.
for instance, in the Spanish flu epidemic of 1918 it was found that young adults were most at risk. in that case, it was concluded to be the robust (or overly robust depending on how you look at it) response of these young adults immune systems that was causing the lethal damage leading to their death.
so it seems the changes in the mortality of the COVID-19 / SARS2 virus is likely to do with some factor in the condition of the older person's immune system also - but of course, we will not have a factual answer for some time yet.
Posted by Marnie (Member # 773) on :
COVID-19 attacks many more organs than just the lungs! Kidneys can take a hit, liver, intestines, heart...though it does increase lung mucus and impact the cilia - negatively.
The majority of COVID-19 ACE2 receptors are - get this - on our tongue. Wear a mask!!!
ACE2 also messes with tryptophan via an enzyme called BOAT2 (a transporter)...down go both ACE2 and availability of Tryptophan.
{Proteases cleave - chop apart proteins (chains of amino acids) so the pathogen can then uses those amino acids for itself. Think of COVID-19 like Edward Scissorhands.) This guy has it absolutely correct:
Hon. Audley Shaw (Minister of Industry, Commerce, Agriculture and Fisheries)
Yesterday at 5:16 PM
There are several dietary flavonoids that ***inhibit corona virus
3CL protease.***
The most studied are luteolin and quercetin, which is found in both *elderberry* and Houttuynia. Food sources of quercetin include onions, apples and many other fruits. Quercetin is presently being studied in China as a drug treatment for COVID-19, based on research initiated at McGill University.
Other flavonoids with potent 3CL protease inhibition in laboratory studies include herbacetin, which is primarily found in ground flax seed (not in flax seed oil but in the husk) and theaflavin gallates, which are abundant in black and puerh tea
Also, All recipes. com has a great healthy cookie to make with wheat flour and ***ground flax seed*** - and chocolate chips of course :-)
There is a difference between regular "flu"viruses (Type A and Type B flu) and coronaviruses.
The Japanese have developed a drug to fight COVID-19 via blocking TMPRSS2 (transmembrane protein receptor serine serine 2) which COVID also needs to replicate. The drug is the "next generation" of a drug already available to treat *pancreatic* inflammation.
(COVID-19 is a BETA coronavirus....think about that implication...)
The new drug is called Nafamostat . Google it along with the word Japan to find the article.