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» LymeNet Flash » Questions and Discussion » Medical Questions » OKAY for those who are messageing me here is what I found on web about my issues

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Author Topic: OKAY for those who are messageing me here is what I found on web about my issues
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On my echo cardiogram I can figure this much out on my own....

1 borderline concentric left ventricular hypertrophy
Hypertension is more prevalent in Black than white men, and is more prevalent in men than women. Furthermore, even controlling for blood pressure, concentric left ventricular hypertrophy, an early structural adaptation of hypertension, is more prevalent in Black than white men, and more prevalent in men than women. Concentric left ventricular hypertrophy is the strongest predictor, other than age, of the cardiovascular morbidity associated with high blood pressure.

SUMMARY: The relative importance of left ventricular (LV) geometry, wall thickness, and mass on the inscription of left ventricular hypertrophy (LVH) on the ECG was examined in 93 patients; 36 had no LVH on the ECG, 10 had borderline voltage for LVH, and 47 had LVH on the ECG. LV cineangiograms in the right anterior oblique projection were analyzed for LV end-diastolic volume, free wall thickness (h), semiminor radius (R), LV mass index (LVMI), and the geometric relations described by h/R, mass/volume, and h * R.

Although mean LVMI was greater in patients whose ECG voltage was either borderline or diagnostic of LVH, increased LVMI also occurred without LVH on the ECG. None of the variables (h, R, h/R or mass/volume) reliably indicated when LVH would be inscribed on the ECG. The product of h * R, however, defined three electrocardiographic groups; all patients with LVH on their ECG had he R >2.6 cm'.

Wall thickening sufficient to result in an increased LV mass did not result in LVH on the ECG unless sufficient concurrent chamber dilatation was present. Thus, a critical geometric relationship resulting from the interplay of wall thickness and chamber dilatation is necessary for LVH to appear on the ECG. This finding is consistent with the solid-angle theory of electrocardiography as it relates to ventricular hypertrophy.

2. normal left ventricular systolic function

nice to know this too!

3.evidence for left ventricular diastolic function

Diastolic left ventricular dysfunction is now increasingly recognized as a condition leading to morbidity, hospitalizations and death. Recent studies suggest that isolated diastolic heart failure occurs in 30-60% of all patients presenting to hospitals with evidence of congestive heart failure (CHF). This review examines the features that make diastolic heart failure a unique clinical entity.

4.continuous wave Doppler reveals no evidence of valvular stenosis
well that is a good thing!

5. color flow Doppler reveals trace tricuspid regurgitation

looking up the regurgitation that is possibly what is causing my pulmonary issue : it will explain why i am so tired and weak.. now to look further into this mess
Tricuspid regurgitation is a disorder involving backward flow of blood across the tricuspid valve which separates the right ventricle (lower heart chamber) from the right atrium (upper heart chamber).

This occurs during contraction of the right ventricle and is caused by damage to the tricuspid heart valve or enlargement of the right ventricle.

Alternative Names:
Tricuspid insufficiency
Causes, incidence, and risk factors:
The most common cause of tricuspid regurgitation is not damage to the valve itself, but enlargement of the right ventricle, which may be a complication of any disorder that causes failure of the right ventricle.

Other diseases can directly affect the tricuspid valve. The most common of these is rheumatic fever, which is a complication of untreated strep throat infections. The valve fails to close properly, and blood can flow back to the right atrium from the right ventricle, and from there back into the veins. This reduces the flow of blood forward into the lungs. The condition affects about 4 out of 100,000 people.

Another important risk factor for tricuspid regurgitation is use of the diet medications called "Phen- fen" (phentermine and fenfluramine) or dexfenfluramine.

BTW I have never been on this drug before ,, never!!

Rarely tricuspid regurgitation can be caused by an unusual tumor called carcinoid. This tumor secretes a hormone which damages the valve.

In the absence of high blood pressure in the lungs (pulmonary hypertension), tricuspid regurgitation is usually asymptomatic. If pulmonary hypertension and moderate-to-severe tricuspid regurgitation coexist, symptoms may include:

active neck vein pulsations
swelling of the abdomen
swelling of the feet and ankles
fatigue, tiredness
decreased urine output
generalized swelling

Signs and tests:
When gently pressing with the hand (palpation) on the chest, there may be a lift produced by the beating of the enlarged right ventricle. Similarly, there may be a pulsation over the liver. The liver and spleen may be enlarged.

Listening to the heart with a stethoscope shows a murmur or abnormal sounds. Ascites (collection of fluid in the abdomen associated with liver disorders) may be present.

Enlargement of the right side of the heart may show on an ECG or echocardiogram.

Pressures inside the heart and lung may need to be measured with Doppler echocardiography or right-sided cardiac catheterization.

Treatment may not be needed if there are few or no symptoms. Hospitalization may be required for diagnosis and treatment of severe symptoms. Underlying disorders should be identified and treated. Surgery to repair or replace the tricuspid valve (heart valve surgery) may be needed.

Expectations (prognosis):
The disorder may correct itself with treatment of underlying disorders, especially treatment of pulmonary hypertension and right ventricular enlargement. Surgical valve repair or replacement usually provides a cure.

worsening of heart failure
endocarditis (heart valve infection)
weight loss, loss of appetite

Calling your health care provider:
Call your health care provider if symptoms of tricuspid regurgitation are present.

I see now why I have a problem with my back and chest pain but why did no one explain this to me.. they say take off as much time to get better but I have not gotten better!!!
so now what?
I am going to show this to my pain management doctor when I get in there since she canceled today cause she wasn't feeling well.

I am going to explain my family situation and the hearing on Monday to her.. that the judge was ready to grant me a determination then.. tell her that forget about going back to work i need to get off permanently and forget the surgery for my back. I dont feel comfortable anymore with the thought of surgery...

I will hang in a pool 24/7 for the pain...along with the fentynal... but this trying to get back to work is going to kill me.... I forfeit I loose on this one cause I can't afford to deal with the struggle, some things are just not worth the fight and this is one of them totally..
whatever will be will be from here in...

Gotta love you people... stop worrying about me.. this explains alot and I am happy to know why my lungs are causing this pain.. and that I can maybe find an answer when I figure out what drug will do the same as the metoprolol that won't cause lung duress ..... LLMD will answer this one I am sure .. teim will provide answers but I do not trust these people locally OKAY .. enough

lets get some sleep on this one tonight...

thanks for all of your concern...
Linda D

"Hatred paralyzes life; love releases it. Hatred confuses life; love harmonizes it. Hatred darkens life; love illuminates it."

Posts: 1389 | From who knows, who cares, but somewhere over the rainbow | Registered: Mar 2003  |  IP: Logged | Report this post to a Moderator

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