This is topic Need scientific doc about tick attachment time in forum Medical Questions at LymeNet Flash.


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Posted by lymeyinok (Member # 5817) on :
 
NEED HELP!! I have a court hearing on Monday about my worker's comp case regarding lyme. I was bitten when on a school out of doors week long trip. My tick was attached for only 36 hours (not the 72-84 hours that is so commonly sited in most literature!) Apparently the doctor for the ins. company has some of those articles in his statement. I am in need of an article stating that the tick does not have to be attached that long --- I know I have seen this in some literature but am unable to find it now that I need it!!!!!!

THANKS!
 
Posted by DJP (Member # 5893) on :
 
Look at the ABC's of Lyme Disease on the LDA's website. The article written by DR. J states that it can be transmitted within hours.
http://www.lymediseaseassociation.org/ABCsLYME.pdf

Also look at "The Basics" - page 16 under
"What should I do if I'm bitten by a tick".
http://www.lymediseaseassociation.org/ABCsLYME.pdf

Most of the literature says 24-36 so even that should help.

Hope those help
 
Posted by cbb (Member # 788) on :
 
I hope that someone will know how to locate the following info:

The Lyme Disease Foundation ( www.lyme.org ) has an International Lyme Disease Conference each year.

If I remember correctly -
Some years ago, a featured speaker was Dr Willy Burgdorfer, discoverer of Borrelia burgdorferi (Bb), the spirochete named for him.
He said that they had found that some ticks have the Lyme spirochetes in the salivary glands when they bite. In those cases, bacteria can be transmitted immediately.

I don't know if a published article includes this info or not.

And,
A well-known LLMD told me that the statements about "48 hours or longer attachment time" were the result of experiments in labs where ticks were removed with proper techniques.
In the "real world", that rarely happens.

Also -
In "Diagnostic Hints & Treatment Guidelines...."
Fourteenth Edition - November, 2002
printed - about 32 pages
www.ilads.org/burrascano_1102.html

Last section:
"Rationale for Treating Tick Bites....
Prophylactic antibiotic treatment upon a known tick bite is recommended for those who fit the following categories....
#3. Persons bitten by a tick capable of transmitting B.burfdorferi, where the tick is engorged, or the attachment duration of the tick is greater than four hours..."

Fifteenth Edition - September, 2005
contains the same info.
To read, go to www.ilads.org
Click 'Articles & Presentations'
www.ilads.org/burrascano_0905.html
 
Posted by mjo (Member # 7876) on :
 
Hope this helps:

Denise Lang's book,"Coping With Lyme" page 8 under Vectors and Hosts section says "tick must be connected four to twenty-four hours..."

But I believe Karen Vanderhoof-Forschner's book "Everything you need to know about Lyme Disease" will serve you better in the section on page 26 Systemic versus Local Infection. One passage explains that ticks which have fallen off another host while they were attached and sucking blood then have the Bb all ready in their salivary glands and can infect immediately.

She also writes that systemically infected ticks can "..tramsmit a pathogen in only a few hours."
 
Posted by welcome (Member # 7953) on :
 
Of note:
I believe I read in an article (maybe PubMed) that the BB pathogen needs to be introduced into the skin. Without this staging area it cannot infect the potential host.

I'm going to try and find this. I'll be right back. [Big Grin]
 
Posted by Kenny from PA (Member # 8007) on :
 
I (too) have read on multiple occasions that the tick only needs to be attached for 24 hours to spread the infection.
 
Posted by welcome (Member # 7953) on :
 
To place an arbitrary time frame on a biological pathogen is idiotic in my opinion. Kind of like claiming a building is "earthquake proof" based on some man made richter number. Any bite that draws blood, from any wild creature, particularly a parasitic one should be considered a dangerous vector. The info below supports the conclusion that if you do acquire the "bulleye" no further testing is necessary, you have acquired Lyme infections. This is the staging and morphing "area" which allows for a fullblown assault on the body.

Page is here

One prominent feature of Lyme disease
is vascular injury (2, 42), and damage to vascular endothelium
results in localized aggregation of activated platelets. Binding
of B. burgdorferi to activated platelets may, therefore, facilitate
tissue colonization by concentrating spirochetes at these
regions. Alternatively, binding of B. burgdorferi to platelets
at the site of inoculation in the skin might provide a foothold
for the establishment of infection-e.g., a niche where the
bacteria can replicate while protected from the host defense
system by a surrounding thrombus. In support of this hypothesis,
in the mouse model it appears that dissemination of
B. burgdorferi from the site of the tick bite is delayed for
several days (3). Alternatively, attachment to platelets may
be more important in the natural vector-reservoir transmission
cycle of B. burgdorferi, for example, by increasing the
local concentration of spirochetes available for ingestion by
an uninfected tick. It is less likely that binding to the hematogenous dissemination of
the spirochete, as activated platelets are not abundant in the
circulation.

Page is here.
Microbial adhesion to and colonization of host
tissue is an early, critical event in an
infection process. In the case of Lyme disease,
host tissue adherence appears to be of importance
during different stages of the disease process.
Initially, during an infected tick's blood meal,
a small number of spirochetes are deposited in
the dermis of the host, where the bacteria appear
to colonize collagen fibers (4, 5). As the
infection disseminates to other tissues, bacteria
may colonize additional extracellular matrix
structures, and host cells may be involved. We
previously showed that adherence of B.
burgdorferi to collagen fibers involved a
specific binding of the spirochete to decorin, a
dermatan sulfate proteoglycan that is associated
with and "decorates" collagen fibers, whereas a
direct binding to collagen could not be
demonstrated (6-9). A dermal route of entry into
the host appears to be important for the
development of disease. Spirochetes administered
intravenously are rapidly and effectively cleared
by Kupffer cells in the liver (10), whereas those
inoculated intradermally consistently establish
infection (11). Perhaps the initial dermal
colonization allows the organism to adapt to in
vivo conditions before blood stream dissemination.

[ 21. October 2005, 12:25 AM: Message edited by: welcome ]
 
Posted by sarahinnewyork (Member # 7179) on :
 
Hi,

I attended a lecture by Dr an New Jersey Dr, Dr G. She said that a tick didn't have to be on very long at all. I know from personal experience that this is true, as I developed Lyme after flicking a tick from my hair. I think that it nipped my finger and that was enough.

Perhaps you can get something in writing from Dr G. She may know what documentation you need to support this...

Best of luck-

Sarah
 


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