did anyone post this previously? i didn't see it in the search engine.
..interesting that they can publish this without having ever studied or proven it so:
~~ snipped ~~
Learning Point Lyme disease is not transmitted by breast milk. The American Academy of Pediatrics RedBook� states that no causal relationship between maternal Lyme disease and pregnancy complications or congenital abnormalities has been conclusively documented.
well, i suppose if there are no real studies being done on this kind of transmission, it's kinda tough to 'conclusively document' it. why state it at all? i see this as beyond misleading. my youngest contracted lyme from my breastmilk, which was dna positive for the organism.
mo
Posted by bettyg (Member # 6147) on :
1 of the members here can prove that wrong.
also, it's in book 2 of PJ LANGHOFF'S IT'S ALL IN OUR HEADS; 1 of 80 stories dealt with this; but i'd have to read all those stories to tell you who!!
The ***DNA from Bb*** has been detected in breast milk, but I'm guessing this is the protective factor:
TGF-[beta] in breast milk
I gotta find where that info is buried in my recent files these past few days...
There it is.
"Programmed cell death, apoptosis, is a natural event in many cells. Overexpression of COX-2 in some cell lines is associated with the expression of Bcl-2, a protein that acts to make cells resistant to apoptosis.
In addition, overexpression of COX-2 is associated with ***decreased*** expression of transforming growth factor-beta (TGF-beta).
TGF-beta is important for transducing signals that inhibit cell growth.
Cells can die in two ways: Through apoptosis, when the cell self-destructs through programmed cell death as a result of "death signals", and through necrosis, which is death from other causes, such as lack of oxygen or toxins.
TGF-β induces apoptosis in numerous cell types. TGF-β can induce apoptosis in two ways: through the SMAD pathway or the DAXX pathway.
More importantly:
TGF-β is believed to be important in regulation of the immune system by CD25+ Regulatory T cell. TGF-β appears to
***block the activation of lymphocytes and monocyte derived phagocytes.***
Once again...no response, no infection.
Bb is counting on us to react. It infects and "dis-arms" our own defense cells.
That is not to say Bb is not transmitted to the infant via other routes (crosses the placental barrier).
Another pathogen can do that because it interferes with Actin-A. Listeria competes with our actin.
The Listeria bacteria use the cellular machinery to move around inside the host cell, by inducing directed polymerisation of actin by the ActA transmembrane protein, thus pushing the bacterial cell around.
"Interestingly, we have also found that the RF Borrelia species tested in this project has a great ability to pass the blood-placenta barrier indicating an effective mechanism to also pass this important physical barrier to an immune privileged site."