Does anyone know what it means when your vitamin D levels are at 177.5, the normal levels are between 0 and 70. I feel like it's the Lyme. I have not seem any improvements from the antibiotics, I feel like it has to do with the vitamin D. Wasn't taken vitamin D pills either. Anyone else experience this? Or know what it means?
Posted by t9im (Member # 25489) on :
Hi Jake:
D3 is converted to Hydroxy 125 by the liver. It is the HD levels that matter. If yours is 177 it is high.
Our daughters was down to 17 from the Lyme, we try to keep it between 60 and 80.
Posted by Jake Hembree (Member # 43187) on :
I'm not sure what the means? Is there a way to bring it down?
Posted by Carmen (Member # 42391) on :
First I would suspect that there was an error with the test.
Posted by Keebler (Member # 12673) on :
- Jake,
You say: "have not seem any improvements from the antibiotics" (end quote)
Are you seeing an ILADS educated lyme literate MD or ND? That is key.
Have coinfections been assessed? Are you gluten-free, dairy-free?
No need to answer, just the basics to check off.
You don't say how long you've been treating. It can take months, or even years to feel better. Sorry. Depends on so much, though. Just be sure that you are with an ILADS doctor and that they have you good support measures, too. Especially liver & adrenal support.
And explore the range of tick borne infections to consider. -
Posted by Keebler (Member # 12673) on :
Topic: NATURAL SLEEP & ADRENAL SUPPORT -
Posted by Lymetoo (Member # 743) on :
Moving to Medical Questions
Posted by Keebler (Member # 12673) on :
- Back to Vitamin D,
Carmen suggests: "First I would suspect that there was an error with the test." (end quote)
Indeed. Test errors happen a lot with Vitamin D testing, actually, especially at certain labs.
A test like that should have been repeated right away to be certain. That is the first step anytime such irregularities show up. The labs should have repeated it without even asking. But then, if they did not, your doctor should have ordered a repeat test.
Make sure before you make any moves. Test again.
But, no matter what you D levels are, if you have an ILADS educated LLMD or LL ND, they should be able to help with this issue, too, or know just who else could help sort it out.
Ask them what lab they suggest to rework the TESTS for Vitamin D levels (it is not just one test, but at least two different ones). -
Posted by Marnie (Member # 773) on :
Ditto...maybe lab error. Was the blood test done after you FASTED?
"Usually you will not need to fast. However, this depends on the laboratory on the testing method used."
"An excess of vitamin D causes abnormally high levels of calcium in the blood. This can severely damage the bones, soft tissues, and kidneys over time. It is ***almost always*** caused by forms of vitamin D that need a doctor's prescription."
Have you been eating lots of oysters? ;-)
Posted by Abxnomore (Member # 18936) on :
Definitely have it retested. That number is extremely high, if it is accurate. If you are taking vitamin D3 stop taking it until you retest. If it's still high, stop it until your levels level off.
Posted by Robin123 (Member # 9197) on :
Which D are you talking about - D3, D4, D25, D-1,25?
Posted by Jake Hembree (Member # 43187) on :
Okay I am looking into a llmd. & the vitamin d was the vitamin calcitriol (1,25 di-oh vitD) which was high. A regular MD just retested and is now in range so it did have to be a error which is good news. Now my regular vitamin d 25 hydroxy is low.
Posted by LisaK (Member # 41384) on :
maybe you can't absorb it?
Posted by Marnie (Member # 773) on :
Vitamin D as it relates to the liver, kidneys and our immune macrophages (watch the spelling changes):
“In the liver, cholecalciferol (vitamin D3) is converted to
calcidiol.
*Part of* the calcidiol is converted
by the kidneys to
calcitriol,
the biologically active form of vitamin D.
Calcidiol is transported to the ***proximal tubules of the kidneys***, where it is hydroxylated at the 1-α position (lower right of the molecule) to form
calcitriol
(aka 1,25-dihydroxycholecalciferol and abbreviated to 1,25(OH)2D).
This product is a potent ligand of the vitamin D receptor (VDR),
which mediates most of the physiological actions of the vitamin."
So…D3-> liver -> makes calcidiol -> some goes to kidneys -> calcitriol which is a ligand (binder) of the vitamin D receptor.
In addition to the kidneys, calcitriol (same ligand of the VDR that the kidneys make) is also synthesized by monocyte-macrophages in the immune system.
When synthesized by monocyte-macrophages, calcitriol acts locally as a cytokine, defending the body against microbial invaders by stimulating the innate immune system."
Now...the BEST form of D maybe D3/K2. Take one a day with a "fatty meal" (the fat soluable vitamins should be taken with something fatty).
Give it time..it will take several MONTHS to work.
The body does not heal fast, it is a slow process to reverse any damage, but it can be done!
K2 is a FORM of vitamin K that looks to ***help keep calcium where it belongs***, not in tissues like the kidneys where an excess of calcium -> kidney stones (also linked strongly to vitamin B6 levels).
Help out your kidneys.
Follow?
Now...while we can and do make that vitamin D ligand (binder), supposedly a precursor to that is cleaved (chopped apart) via a tryptophan containing enzyme that Bb supposedly secretes called "nagalase". Ultimately it is believed this prevents our macrophages from working fully since vitamin D loses its ability to lock on.
That enzyme (naglase) in research...in a 5% dextrose solution is capable of changing blood type A and/or B to blood type O (removing antigens i.e., proteins).
I documented under a heading "nagalase GcMAf"
"Herpes simplex virus type 1 glycoprotein gC is an
immunoevasin
that
inhibits complement activation by binding complement C3b."
Are we still reacting to p8...HTVL-1 (= retroviral protein) that Bb picks up in the tick's saliva? Does that retroviral protein have Gc too?