This is topic Breast cancer possible cure in forum Medical Questions at LymeNet Flash.


To visit this topic, use this URL:
https://flash.lymenet.org/ubb/ultimatebb.php/topic/1/135311

Posted by Marnie (Member # 773) on :
 
We know Bb can attach to and even invade HeLa cells (HPV triggered cancer cells) and Bb shares lipids (proteins) with those cells.

Tumour cells are frequently in shortage of polyunsaturated fatty acids.

Docosahexaenoic acid (DHA), a well-known ligand of the PPAR family, has been shown to reduce tumour proliferation in lung tumour cell cultures

https://www.hindawi.com/journals/ppar/2016/2308249/


DHA + all trans retinoic acid.


Apoptosis = cell death

J Cancer Res Ther. 2016

The combined effects of all-trans-retinoic acid and docosahexaenoic acid on the induction of apoptosis in human breast cancer MCF-7 cells.

CONCLUSION:

We conclude that the

combination of ATRA and DHA at the well-balanced proportion

***may be effective in cancer cell apoptosis.***

Further studies provide details about the potential synergistically effects of combination treatment of ATRA and DHA in growth inhibition and differentiation of human mammary cancer cells.


PMID:27072238


***All-trans retinoic acid (ATRA) downregulates MMP-9 ***

by modulating its regulatory molecules.

https://www.ncbi.nlm.nih.gov/pubmed/20421725


Read more about MMP9 here:

https://en.wikipedia.org/wiki/MMP9

See the mention of Doxy in the above?!


Tretinoin, also known as all-trans retinoic acid (ATRA), is medication used for the treatment of acne and acute promyelocytic leukemia. Wiki.

Conclusions.: Our results show that RA (retinoic acid) activation of the p38 intracellular pathway was essential for its

early induction of apoptosis

and partially responsible for promoting differentiation.

***DHA prevention of this apoptosis***

suggests that RA effects during early development

must be counterbalanced by survival factors

to prevent photoreceptor death, in an interplay that might help to establish the final number of photoreceptors.

http://iovs.arvojournals.org/article.aspx?articleid=2189053

Bear with me...

The WFL while hunting for food at night loves (and needs) to eat crickets. Crickets contain a lot of critical nutrients that activate rhodopsin in eye rods in order to see at night. When Bb loaded ticks feed on that lizard, Bb is killed.

"Crickets are also important sources of numerous necessary nutrients such as the 8 essential amino acids, vitamin B12, riboflavin,

***the biologically active form of vitamin A

(retinol, retinoic acid, and retinaldehyde)***

and several minerals such as Iron and Calcium."

http://webcache.googleusercontent.com/search?q=cache:Ig1_ZzKjt5IJ:https://cricketpowder.com/nutrition/

***Vitamin A activates rhodopsin*** and sensitizes it to ultraviolet light.

PMID: 22192505

Repeating...crickets contain vitamin A nutrients required to activate rhodopsin so the WFL can see in the dark (rhodopsin captures any available light photons).


The chromophore imparting a

*** red color to rhodopsin***

was a vitamin A-derived compound called 11-cis-retinal.

Moreover, he found photons striking this chromophore could induce a change in its configuration from an 11-cis, through a series of photointermediates,

***to an all-trans state...***

(Rhodopsin + light-photons = "all trans" = ATRA)


Retinol is produced in the retina from Vitamin A, from dietary beta-carotene.

Isomerization of 11-cis-retinal
***into all-trans-retinal by light ****
induces a conformational change (bleaching) in opsin,

continuing with metarhodopsin II,

which activates the associated G protein transducin

and triggers a Cyclic Guanosine Monophosphate, second messenger, cascade…

Wiki

Metarhodopsin II activates the G protein transducin (Gt) to activate the visual phototransduction pathway.

When transducin's α subunit is bound to GTP, it activates cGMP phosphodiesterase.

cGMP phosphodiesterase hydrolyzes cGMP

***(breaks it down).***

cGMP can no longer activate cation channels

The "English" translation =

The WFL's diet of crickets contains a lot of active vitamin A which activates rhodopsin which captures light photons -> ATRA -> metarhodopsin II -> transducin -> cGMP phosphodiesterase =

Degradation of cGMP causes

sodium channels to close!!! Wikipedia.

The above is how, I think, the WFL is capable of destroying Bb.

Bb is very, very dependent on Na - sodium:

The authors discussed that this spectrum of attractants/repellents, in addition to the fact that

NaCl was needed for motility, makes

it likely that B. burgdorferi uses chemotaxis to direct its movement through tissues.

The physiological conditions of the interstitial fluid meet the requirements for B.

burgdorferi motility (high NaCl and pH 7.6).


http://webcache.googleusercontent.com/search?q=cache:5u7JX3LEB20J:http://www.horizonpress.com/jmmb/v/v2/v2n4/04.pdf

**********

While ATRA downregulates MMP9, so does...


HBOT also looks to also downregulate MMP9 ;-)

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4658863/

HBOT plus DHA? Remember the breast cancer treatment included ATRA which lowers MMP9 AND DHA.

(DHA is "structural" and is a component of our cell walls. Whereas EPA is more functional - reducing inflammation.)

More about DHA here - see problems with deficiency:

https://en.wikipedia.org/wiki/Docosahexaenoic_acid

Not all DHA sources are the same:

Rats fed KO (krill oil) had significantly lower DHA digestibility and brain DHA incorporation than SO (salmon oil) and TO-fed (tuna oil) rats.

https://lipidworld.biomedcentral.com/articles/10.1186/1476-511X-10-179

The DHA diet also

counteracted

the FPI-***reduced manganese superoxide dismutase*** (SOD) and Sir2 (a NAD+-dependent deacetylase).

http://online.liebertpub.com/doi/abs/10.1089/neu.2011.1872

IF HBOT upregulates ROS, those ROS might ->

A metabolite of lipoxygenase-mediated

***oxidation of DHA; ***

activates Nrf2-dependent antioxidant gene expression,

acts as a ***PPARγ agonist***

(EC50 ~200nM),
and inhibits pro-inflammatory cytokine and nitric oxide production at biological
concentration ranges

http://webcache.googleusercontent.com/search?q=cache:TIUZcMT4EyYJ:https://www.cedarlanelabs.com/Contents/Files%3FfilePath%3Dpdf%255CCayman%2520Chemical%2520-%2520Metabolism%2520Bro chure.pdf

So DHA oxidized -> upregulation of PPARy.

"Along with this are secondary effects due to the gram negative nature of Bb bacteria. Gram negative bacteria have a lipopolysaccharide cell wall “LPS”.

The LPS are neurotoxins which have a low molecular weight which affect the whole body through the following mechanisms:

1. Disrupts sodium and calcium channel receptors

2. Attacks enzyme reactions involving glucose processing at the cellular level

3. Manufactures renegade fatty acids impairing membrane function.

4. Stimulates phospholipase A2 (PLA2) which prematurely uncouples essential fatty acids from cell membranes

***5. Impairs the function of PPAR gamma which affects changes in DNA function and gene switching.*** - from The Detoxx Book by John Foster M.D. et al."

http://lyme.startpagina.nl/forum/topic/1028313/richard-bierman-on-lyme-disease/

2016...will surprise you

http://tinyurl.com/zzd5g7q

I suspect lyme disease can trigger breast cancer

in genetically susceptible persons.

In addition to BRCA1 and BRCA2, five other genes can be considered well established breast cancer susceptibility genes.

[ 01-03-2017, 01:38 AM: Message edited by: Marnie ]
 
Posted by patches10025 (Member # 20983) on :
 
Have you heard of Dr. Morse in Port Charlotte?
 
Posted by bluelyme (Member # 47170) on :
 
I love how your brain works marnie , georgia hunter on hw and you could be a force to be reconed with ...i was always wondering about my photosensitivity and that info about wfl maybbe spot on ..just amazing .thank you

dr m thinks lyme can be cured with pinapple?

[ 01-03-2017, 01:42 AM: Message edited by: bluelyme ]
 
Posted by Marnie (Member # 773) on :
 
I am not familiar with Dr. M. (names are not permitted on this site).

He is an interesting person with an unusual perspective.

Hum...pineapple...


Furthermore, bromelain inhibits the invasiveness of human cancer cells by

suppressing matrix metalloproteinase (MMP)-9 expression

through inhibiting activator protein 1 (AP-1)

and NF-κB signaling pathways.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4998156/

In a mouse model, MMP9 ***deficiency*** resulted in

resistance to endotoxin shock,

suggesting that MMP9 is important in sepsis.

Doxycycline suppresses the growth of aortic aneurysms through its inhibition of MMP9.

Wikipedia

Bromelain digests proteins.

Web MD's "take":

http://www.webmd.com/vitamins-supplements/ingredientmono-895-bromelain.aspx?activeingredientid=895&activeingredientname=bromelain

[ 01-03-2017, 01:06 AM: Message edited by: Marnie ]
 
Posted by Robin123 (Member # 9197) on :
 
Very interesting. Noni juice and grapeseed extract also fight MMP9 inflammation.
 


Powered by UBB.classic™ 6.7.3