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Posted by lookin4answers (Member # 4974) on :
 
http://www.eurekalert.org/pub_releases/2005-05/nymc-srn042905.php

Study reveals new data on how lyme disease is spread

New York Medical College researchers publish new findings on the spread of lyme disease bacteria

The results of a five-year study, published this week in the Annals of Internal Medicine by researchers at New York Medical College, reveal intriguing new data on the spread of the Lyme disease bacteria through the blood stream. The ability to find the Lyme spirochete--the tick-borne agent responsible--in the blood is itself an achievement because existing methods of culturing blood were not sensitive enough to detect its presence until College researchers developed a new technique, which they used in the study.
Leading the study was Gary P. Wormser, M.D., professor of medicine, director of the Division of Infectious Diseases and vice chairman of the Department of Medicine. "If Lyme disease stayed in the skin it would be a completely different and rather inconsequential infection--but it doesn't," Dr. Wormser explained. "The causative agent of Lyme disease can spread from its entry point at the tick bite site through the blood to distant sites such as the brain, heart, and joints. This study answers questions that have never been answered before and raises others that will likely stimulate future studies on Lyme disease."

The study followed 213 initially untreated adults with erythema migrans, the tell-tale bull's eye rash that is the most common clinical feature of Lyme disease, to determine when does blood stream invasion occur, how many patients experienced blood stream invasion and who is most vulnerable. Blood stream invasion occurred in 93 (43.7 percent) of patients in the study, who also were more often symptomatic (89.2 percent vs. 74.2 percent) and more likely to have multiple erythema migrans lesions (41.9 percent vs. 15.0 percent) than the 120 patients without blood stream invasion. However, some patients had no tell-tale symptoms at all, making the presence or absence of blood stream invasion impossible to predict with certainty based on clinical features alone. Younger patients and those with a prior history of Lyme disease were somewhat protected.

Dr. Wormser and his colleagues concluded that, "The high rate, early onset, and prolonged duration of risk for blood stream invasion probably explain why untreated patients with erythema migrans often develop complications distant from the tick bite location. Older patients without a past episode of Lyme disease are at particular risk."

 


Posted by Lymetoo (Member # 743) on :
 
Since Wormser is a big WORM I have my doubts that anything good will come of this study....but we can hope.

So what did they do ... experiment on people and not treat them until they were finished with them? They probably gave them 3 days of abx and sent them on their way!

------------------
oops!
Lymetutu

 


Posted by Lymerayja (Member # 6839) on :
 
Hey there Lookin4answers, I don't think we'll find many answers from Wormser and his wormy crew at NYMC.

Hard to imagine that the man who brought us the ALDF and the IDSA guidelines (along with his filthy friends) is going to come up with anything to help Lyme patients.

They refer to the "initially untreated" people with EM that he collected for his study - did it ever occur to him that those people were probably untreated because of the extreme ignorance and disinfo that he and the rest of the Steerites have promoted among the medical profession???

Here's a little piece about the formation of the IDSA Lyme committee, the folks who brought you "Chronic Lyme does not exist", starring Wormser and other bastards like Nadelman, Rahn etc.

You can also print this out for times when you are stuck in a traffic jam or stuck in bed or just bored and amuse yourself with a little game of "How many insurance companies can you spot?" and "match the Steere camp doc with the conflict of interest".

Happy reading.

Lisa

From the LymeLight Newsletter of the Lyme Disease Foundation:

"...When Donta learned the [IDSA] guidelines were going to be published, he said he voiced his desire to make changes to its late-stage section. Rather than consider his suggestions, Dr. Donta said committee member Robert Nadelman, MD, informed him that unanimity was not required for publication, and Donta's name was removed from the protocol without explanation.

For reasons that remain unclear, the protocol was taken from committee chair Benjamin Luft, who Donta believes wanted to make changes to its late stage section, and given to Dr. Gary Wormser, an insurance consultant (Aetna) who took over as IDSA chair during the reorganization of the committee. Dr. Donta previously told LymeLight he believed the IDSA meetings "were based on politics, not science" and that when the guidelines were published, they would be "an embarrassment to the Society." Dr. Luft did not return messages to discuss the protocol.
Documents obtained by the LDF support Donta's conclusion.

In a October 1995 e-mail, former NIH Lyme Disease Program Officer Edward McSweegan (who was removed from his position), tells new NIH Lyme Disease Program Officer Phil Baker that the American College of Physicians (ACP) and IDSA are currently working on Lyme disease diagnostic and treatment protocols.


McSweegan says Dan Rahn, who was in charge of CDC-funded ACP protocol and an IDSA member for the LD protocol, is "supposed to be talking with" IDSA chair Benjamin Luft, MD to "avoid duplication incompatibility." Rather than "copy the wheel," (presumably by writing a third identical protocol), McSweegan suggests NIH find a way "to support the current efforts or provide some kind of endorsement for them."


Adding further suspicion of collusion to establish one specific protocol for LD diagnosis and treatment is a portion of the book "Lyme Disease" authored by IDSA-committee membr Dan Rahn,M.D., M.P.H., and Yale physician Janine Evans, M.D., and the American College of Physicians.

Page 199 of the book states: "Nationwide, efforts are well underway to limit practice variation, reduce unnecessary health care expenditures, and measure and improve patient outcomes."


Not only might this pertain to avoiding "duplication incompatibility" on authoritative LD protocols, but also one must wonder its relevance to the nationwide harassment of physicians by state health departments against doctors who deviate from "authoritative" protocols such as the IDSA's and treat beyond their recommendations.

Reached at his office at the Medical College of Georgia (MCG), Dr. Rahn, Chair of the MCG-Blue Cross/Blue Shield Center of Health Care Improvement, said no effort was made to coordinate protocols..."

quote:
Originally posted by lookin4answers:
http://www.eurekalert.org/pub_releases/2005-05/nymc-srn042905.php

Study reveals new data on how lyme disease is spread

New York Medical College researchers publish new findings on the spread of lyme disease bacteria

The results of a five-year study, published this week in the Annals of Internal Medicine by researchers at New York Medical College, reveal intriguing new data on the spread of the Lyme disease bacteria through the blood stream. The ability to find the Lyme spirochete--the tick-borne agent responsible--in the blood is itself an achievement because existing methods of culturing blood were not sensitive enough to detect its presence until College researchers developed a new technique, which they used in the study.
Leading the study was Gary P. Wormser, M.D., professor of medicine, director of the Division of Infectious Diseases and vice chairman of the Department of Medicine. "If Lyme disease stayed in the skin it would be a completely different and rather inconsequential infection--but it doesn't," Dr. Wormser explained. "The causative agent of Lyme disease can spread from its entry point at the tick bite site through the blood to distant sites such as the brain, heart, and joints. This study answers questions that have never been answered before and raises others that will likely stimulate future studies on Lyme disease."

The study followed 213 initially untreated adults with erythema migrans, the tell-tale bull's eye rash that is the most common clinical feature of Lyme disease, to determine when does blood stream invasion occur, how many patients experienced blood stream invasion and who is most vulnerable. Blood stream invasion occurred in 93 (43.7 percent) of patients in the study, who also were more often symptomatic (89.2 percent vs. 74.2 percent) and more likely to have multiple erythema migrans lesions (41.9 percent vs. 15.0 percent) than the 120 patients without blood stream invasion. However, some patients had no tell-tale symptoms at all, making the presence or absence of blood stream invasion impossible to predict with certainty based on clinical features alone. Younger patients and those with a prior history of Lyme disease were somewhat protected.

Dr. Wormser and his colleagues concluded that, "The high rate, early onset, and prolonged duration of risk for blood stream invasion probably explain why untreated patients with erythema migrans often develop complications distant from the tick bite location. Older patients without a past episode of Lyme disease are at particular risk."



 


Posted by Lymetoo (Member # 743) on :
 
quote:
Originally posted by lookin4answers:

The study followed 213 initially untreated adults with erythema migrans, the tell-tale bull's eye rash that is the most common clinical feature of Lyme disease.....

this is the part i was referring to
 


Posted by lou (Member # 81) on :
 
I continue to marvel at how the Steere camp churns out research that either proves we don't need treatment beyond the minimal, regardless of the stage of disease, documents the obvious, or otherwise does nothing to help us. This study shows that lyme circulates to distant places. Well, gee whiz, what a breakthrough!!! Give those boys some more NIH grants.

Their research program is designed to give the illusion of progress, without actually moving forward.

Wormser is a creep, will produce no research that helps us, and has done considerable to suppress anything or anyone that does help.

Now are they going to use their new technique of culturing to help diagnose us? Fat chance.

[This message has been edited by lou (edited 02 May 2005).]
 


Posted by lookin4answers (Member # 4974) on :
 
Well, I did not do my research again. My mom said I would catch some flack!!

My bad...did not mean to get anyone in an uproar. She also said he was a WORM!!

TuTu, I apparently read over it too quickly and I agree with you all.

Sorry,
ALF
 


Posted by Lymetoo (Member # 743) on :
 
quote:
Originally posted by lookin4answers:
My bad...did not mean to get anyone in an uproar. She also said he was a WORM!!



No problem, Amanda....live and learn! Gave us a chance to express our displeasure with the WORM. You helped educate a few people!

------------------
oops!
Lymetutu

 


Posted by Lymerayja (Member # 6839) on :
 
No need to apologise, ALF, it's easy to get fooled by the Worm and his mangy colleagues when they present studies that appear to tally with what we intuitively know. It's only by knowing the past history of these con artists, that we know they can't be trusted.

One of the things Gary the Worm Wormser is famous for is chairing the IDSA committee that decided the chronic Lyme - officially - does not exist.

I'd like to share with you gals and guys something that scientist Kathleen Dickson wrote today on another group, in a thread about Wormser's paper. In particularly note the sentences that I've marked with stars.
Lisa

Barbour published about that in 1986.

It was the state of the art in 1975.

The only reason we can't find "Lyme"
in the blood, is because **we use primers
intended to not find it**. Klempner refused
to tell me which primers he used in his
Chronic Lyme "study."

*****BSK does not support all borrelial
spirochetes, and that is intentional.*****

This we also learned from the pharming
of OspA out of E. coli.

It isn't possible that these guys are
so ignorant. "The Biology of Borrelia
Species" had over 250 references in it
some going back to the 1920s.

And that was published by Barbour in
1986. The report by Steere that same
year on what is the serology of Lyme,
looked like Steere knew absolutely
nothing about spirochetes. Or for
that matter, infections.

Kathleen>


quote:
Originally posted by lookin4answers:

My bad...did not mean to get anyone in an uproar. She also said he was a WORM!!

ALF


 


Posted by Ann-OH (Member # 2020) on :
 
A few years ago, another study showed that Lyme spirochetes could not survive in oxygenated areas and so went into tissue as quickly as possible and actually was able to disseminate through tissue. Since blood was very oxygenated, they could not survive in blood for very long. That is partly why spirochetes are rarely found in blood.

The theory beyond Hypobaric Oxygen treatment is based on the assumption that borrelia burgdorferi cannot survive when in a high oxygen environment.

Doesn't Wormser's work fly in the face of logic?

Ann - OH
 


Posted by Lymerayja (Member # 6839) on :
 
Do you know where I can find this study, Ann?
I'm a little bit skeptical. After all, when an an uninfected tick first acquires Lyme, say from an infected deer, the Bb is swimming in blood and must be there for a reasonable length of time prior, unless it had some mechanism of knowing there's a hungry tick nearby

I'm not skeptical that oxygen damages Bb, but the oxygen in the blood is in a chemically bound form, bound to haemoglobin. Also, there are a number of researchers who claim to have found the Bb in the blood and/or cultured it (even though the Steere camp discredits them all).


Lisa

quote:
Originally posted by Ann-OH:
A few years ago, another study showed that Lyme spirochetes could not survive in oxygenated areas and so went into tissue as quickly as possible and actually was able to disseminate through tissue. Since blood was very oxygenated, they could not survive in blood for very long. That is partly why spirochetes are rarely found in blood.

The theory beyond Hypobaric Oxygen treatment is based on the assumption that borrelia burgdorferi cannot survive when in a high oxygen environment.

Doesn't Wormser's work fly in the face of logic?

Ann - OH



 


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