Marnie
Frequent Contributor (5K+ posts)
Member # 773
posted
"cooler objects
emit photons (= pure energy, no "charge")
at longer wavelengths"
Infected cells maybe emitting energy (losing photons).
To counter this loss of energy, the body responds by opening channels.
The TRPM8 channel is activated:
Voltage- and Temperature-Dependent Activation of TRPM8.
The TRPM8 channel is activated not only by
decreasing temperature
but also by
membrane depolarization.
Depolarization typically *results from* the influx of positively charged ions (such as sodium or calcium) into the cell.
Ion channels open or close in response to depolarization or hyperpolarization.
Repeating...when Na and Ca go in the cell, this looks to cause depolarizion of the cell membrane.
It appears this triggers glutamate, not acetylcholine, release (see my post today about Bb's toxins)
Bb appears to have a "taste for" glutamate (= glutamic acid) which is one of our major brain "workhorses" and is the "accelerator". GABA, the "brakes" comes FROM glutamate.
Glutamate -> GABA -> GABA A and GABA C (fast inhibition, chloride channels) and GABA B (slow inhibition, not a chloride channel).
To fully activate GABA B, we need TWO helpers: Gi and Go...
Now...to PREVENT lyme, Frontline (for DOGS!) appears to block the chloride channels, but does not block GABA B which is NOT a chloride channel.
Simplified:
It appears Bb "chills out" the cells it infects and the body responds by sending in NaCl and CaCl to "de-ice".
When Na and Ca are briefly in the cells, this looks to trigger glutamate release.
(MgCl is also a de-icer.)
Bb then wants Ca OUT...which is what this channel does.
Bb is very dependent on Na.
The flow of ions into and out of our cells is happening very very very fast.
Ancora Imparo!
(Trying hard)
Posts: 9424 | From Sunshine State | Registered: Mar 2001
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