Tincup
Honored Contributor (10K+ posts)
Member # 5829
posted
Interesting to note-
*In the 1970's Bumsteere only found 25% of Lyme patients had a rash. Now the standard line being touted by the IDiots is 80%-90% have a rash. They KNOW that isn't true and KNOW they are missing cases because of that figure- and are misleading doctors who WE must deal with.
*The IDiots documented that the rash typically lasted about 3 weeks, yet other symptoms were ongoing or increasing. Today their theory is to treat with 3 weeks of antibiotics- KNOWING the rash will disappear on its own (treatment or not) and KNOWING the disappearance of a rash should not be used as being a "curative" situation, but is being used against patients.
The fact the rash was gone and the person was supposedly "cured" (according to the IDiots) was used to determine success in clinical trials, used to develop tests and then both were used in the vaccine trials to determine a supposedly "successful" outcome.
Check out a couple of the early studies- they are interesting and will help explain why YOU are sick today and show you the IDiots KNEW better and are still promoting the bad information to cover their bee hinds.
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Arthritis Rheum. 1977 Jan-Feb;20(1):7-17.
Lyme arthritis: an epidemic of oligoarticular arthritis in children and adults in three connecticut communities.
Steere AC, Malawista SE, Snydman DR, Shope RE, Andiman WA, Ross MR, Steele FM.
An epidemic form of arthritis has been occurring in eastern Connecticut at least since 1972, with the peak incidence of new cases in the summer and early fall. Its identification has been possible because of tight geographic clustering in some areas, and because of a characteristic preceding skin lesion in some patients.
The authors studied 51 residents of three contiguous Connecticut communities -- 39 children and 12 adults -- who developed an illness characterized by recurrent attacks of asymmetric swelling and pain in a few large joints, especially the knee.
Attacks were usually short (median: 1 week) with much longer intervening periods of complete remission (median: 2.5 months), but some attacks lasted for months.
To date the typical patient has had three recurrences, but 16 patients have had none. A median of 4 weeks (range: 1-24) before the onset of arthritis, 13 patients (25%) noted an erythematous papule that developed into an expanding, red, annular lesion, as much as 50 cm in diameter.
Only 2 of 159 family members of patients had such a lesion and did not develop arthritis (P less than 0.000001). The overalll prevalence of the arthritis was 4.3 cases per 1,000 residents, but the prevalence among children living on four roads was 1 in 10. Six families had more than 1 affected member.
Nine of 20 symptomatic patients had low serum C3 levels, compared to none of 31 asymptomatic patients (P less than 0.005); no patient had iridocyclitis or a positive test for antinuclear antibodies.
Neither cultures of synovium and synovial fluid nor serologic tests were positive for agents known to cause arthritis.
"Lynne arthritis" is thought to be a previously unrecognized clinical entity, the epidemiology of which suggests transmission by an arthropod vector.
PMID: 836338 [PubMed - indexed for MEDLINE]
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Ann Intern Med. 1977 Jun;86(6):685-98.
Erythema chronicum migrans and Lyme arthritis. The enlarging clinical spectrum.
Thirty-two patients with the onset of erythema chronicum migrans, Lyme arthritis, or both in mid-1976 were studied prospectively.
The skin lesion (24 patients) typically lasted about 3 weeks, beginning as a red macule or papule that expanded to form a large ring with central clearing.
Associated symptoms ranged from none to malaise, fatigue, chills and fever, headache, stiff neck, backache, myalgias, nausea, vomiting, and sore throat. Three patients had been bitten by ticks at the site of the initial lesion 4 to 20 days before its onset.
Nineteen patients suddenly developed a monoarticular or oligoarticular arthritis 4 days to 22 weeks (median, 4 weeks) after onset of the skin lesion; eight developed arthritis without a preceding skin lesion.
Seven of these 27 experienced migratory joint pains. Arthritis attacks, most commonly in the knee, were typically short (median, 8 days) but sometimes persisted for months.
Other manifestations included neurologic abnormalties, myocardial conduction abnormalities, serum cryoprecipitates, elevated serum IgM levels, and elevated erythrocyte sedimentation rates.
The diagnostic marker is the skin lesion; without it, geographic clustering is the most important clue.
Tincup
Honored Contributor (10K+ posts)
Member # 5829
posted
Here Bumsteere called the Lyme infection a virus and many people were never given antibiotic treatment and suffered greatly. We had a lot of suicides at that time and for many years afterward.
Hosp Pract. 1978 Apr;13(4):143-58. Lyme arthritis: a new clinical entity. Steere AC, Hardin JA, Malawista SE.
Named for the Connecticut town where the first identified cases occurred in 1972, this disorder has since been found elsewhere and may be caused by a virus transmitted by ticks.
Attacks are often preceded by erythema chronicum migrans and are seldom prolonged, though they may recur.
Symptomatic treatment only is advised, except in the rare instances of severe neurologic complications or myocardial conduction abnormality.
Tincup
Honored Contributor (10K+ posts)
Member # 5829
posted
Sad how the IDiots purposely left people untreated just to study them and use that information to pad their pockets.
And they admitted there were still more symptoms developing in the treated patients - indicating the treatment wasn't a cure! It only made the rash duration shorter- a rash they knew would go away on its own anyway.
Reminds me so much of the Tuskegee experiments. SHAME, SHAME!!
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Ann Intern Med. 1980 Jul;93(1):1-8. Antibiotic therapy in Lyme disease. Steere AC, Malawista SE, Newman JH, Spieler PN, Bartenhagen NH.
We studied antibiotic efficacy in 113 patients with erythema chronicum migrans, the first manifestation of Lyme disease.
Erythema chronicum migrans and its associated symptoms resolved faster in patients given penicillin or tetracycline (median duration, 4 and 2 days, respectively) than in untreated patients (10 days; P less than 0.001 and P = 0.005, respectively). Erythromycin had no significant effect.
**** Although the frequency of subsequent neurologic and cardiac abnormalities was similar in all four groups, significantly fewer patients given penicillin developed arthritis than did untreated patients (P = 0.001). ****
Among 15 patients with arthritis who were followed for at least 29 months, the total duration of joint involvement was shorter in penicillin-treated patients (median, 4 weeks) than in untreated patients (17 weeks; P = 0.019).
Although the clinical manifestations of the disease may fluctuate in frequency from year to year and influence apparent antibiotic effect, we conclude that penicillin therapy shortens the duration of erythema chronicum migrans and may prevent or attenuate subsequent arthritis.
TerryK
Frequent Contributor (5K+ posts)
Member # 8552
posted
The IDSA assumes that nothing but their own research is valid and then only when it suits their needs.
Another example: In "Cure Unknown", the author states that only 4 of the 20 strains that can cause rash actually cause dissemminated disease.
The author states that all the studies that were based on the rash assumed that all of those with a rash were infected when a significant number of strains won't cause disseminated infection.
The implications of this is far reaching given that the foundation of their studies are based on a severely flawed premise.
Soooo, if a person had a rash, was treated and recovered quickly with no residual effects does this mean lyme is easily cured or they never had lyme to begin with?? Many more questions along that line and more should be asked by the IDSA.
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TerryK
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1: J Emerg Med. 2007 Oct 16; [Epub ahead of print]
An Update on the Diagnosis and Treatment of Early Lyme Disease: "Focusing on the Bull's Eye, You May Miss the Mark"
Stonehouse A, Studdiford JS, Henry CA. Jefferson Medical College, Thomas Jefferson University, Philadelphia, Pennsylvania.
To confidently diagnose and treat Lyme disease, the clinician must first understand the natural history of this disease, especially its protean early manifestations.
Emergency physicians, primary care physicians, and other providers need to be vigilant in terms of the timely recognition of erythema migrans (EM), the unique marker of early localized stage 1 disease.
The classic EM, originally described as a slowly expanding bull's eye lesion, is now recognized to be present in only the minority of cases (9%); the dominant morphologic lesion of EM is now recognized to be the diffusely homogenous red plaque or patch, which occurs in over 50% of cases.
This update will define the current morphologic features of early Lyme disease, the indication for serologic studies, and the most recent treatment guidelines, including therapeutic pitfalls.
PMID: 17945460 [PubMed - as supplied by publisher]
Posts: 6286 | From Oregon | Registered: Jan 2006
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Tincup
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Terry- Yep, you are right! And look at this line...
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