Researchers - heads up. Look closely at Mastic gum (capsules and the actual gum)as it relates to H. Pylori AND candida. Used for YEARS (Greeks).
H. Pylori uses Fe-SOD and catalase to avoid the oxidation of its lipoproteins. It also triggers the neutrophils to release elastase - lung implications! AND uses urease to breakdown urea to ammonia and CO2.
"...through the Borrelia burgdorferi production of urease enzyme!"
That is one heck of a lot of ammonia if H.Pylori and Bb are cohorts!
If H.Pylori is knocked off (Augmentin) and -> severe stomach pains due to the beta lactamase inhibitor - 50% of HP contains toxins which do a "number" on the stomach cells. May need to counter quickly with activated charcoal (one capsule).
Resistant candida can be hit with diflucan + berberine HCL (synergy).
[ 10-16-2015, 12:50 PM: Message edited by: Marnie ]
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sammy
Frequent Contributor (5K+ posts)
Member # 13952
posted
What about the candida strains that are inherently resistant to diflucan like Candida Glabrata? Or Candida Krusei? Is berberine enough to make them susceptible again?
What about with other drugs like Itraconazole? Berberine combine with that to overcome resistance?
Adding to ask, any idea how much berberine would be needed as a preventative? Or for combo treatment? This could change medicine if doctors paid attention.
People like me with immune deficiencies are so susceptible to opportunistic infections. Sadly, current medical treatments are not that good for fungi. Any advances in research should be taken seriously and watched.
So to me, this is super interesting.
(Please forgive me as I'm very tired, hopefully I'm not misinterpreting anything. )
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Marnie
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posted
Go to the pubmed abstract linked...Berberine HCL and difulcan for resistant candida = they work in synergy.
Resistant...those nasty export pumps...
The IgY in "hyperimmune egg (yolk) powder...
"chicken IgY against human B-CK"
A good thing or bad? Warning...technical.
B-CK...
We analyzed the role of ***brain-type creatine kinase (CK-B)***, an enzyme involved in high-energy phosphoryl transfer.
We demonstrate that endogenous CK-B in macrophages is mobilized from the cytosolic pool and coaccumulates with F-actin at nascent phagosomes.
Live cell imaging with XFP-tagged CK-B and ß- actin revealed the transient and specific nature of this partitioning process.
Overexpression of a catalytic dead CK-B or CK-specific cyclocreatine inhibition caused a significant reduction of actin accumulation in the phagocytic cup area, and reduced complement receptor-mediated, but not Fc-yR-mediated, ingestion capacity of macrophages.
Finally, we found that inhibition of CK-B affected phagocytosis already at the stage of particle adhesion, most likely via effects on actin polymerization behavior.
We propose that
CK-B activity in macrophages
contributes to complement-induced F- actin assembly events in
Bb residing in macrophages (viable keets or remnants of Bb that continue to exist via CK -B providing the macrophages with enough ATP.
Knock them off...inhibit cytosol CK-B and macrophages are kapoot?
Mastic gum...
More interesting, the mastic tree extracts were also able to destroy harmful fungi such as the common Candida albicans, as well as Candida parapsilosis, Torulopsis glabrata and Cryptococcus neoformans.
I'll let you know. I got it in the mail today.
Also ordered IgY powder.
They gave chickens a vaccine against many gastric pathogens and they made antibodies to them...IgY went up in the eggs - yolk portion.
I'm dealing with H. Pylori triggered bronchitis. I knew I should not have take the steroid dose pack...
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