No wonder I always describe some of my Neurological symptoms like that of someone who has Alzheimers (AD).!!

I wonder what Lyme does not effect in a direct or direct manner!!!!!!!!

In one of his papers, Bransfield spoke of white matter and grey matter memory, AD, and

neuroborreliosis.

The memory itself is contained in the grey matter(cortex of the brain), and associations with a given memory is a white matter(subcortical brain structures) thing.

when the grey matter is gone, memory itself is gone. this characterizes AD

Lyme attacks white matter, predominantly, causing subcortical degradation.
the association with the memory being affected, causes no, or slow retrival of the memory(?)my guess.

see Bransfield et.al. for better description.
as I have a retrieval problem right now.

this might be an ex.:
i had an advanced exam. one of the first and easiest problems had the symbol for the square root (a radical sign), underneath which was a lot of "stuff" that looked-nasty,but really wasn't, but I immediately knew how to solve it.
However, I could not, for the life of me figure out how to get rid of the radical enclosing the nasty-looking stuff, in order to solve the nasty.

interpretation.

an association of the symbol for the radical with a memory of '1/2'. associating the pictorial rendition of the square root with its numeric counterpart.

if its not this, then its something close an example.

the 'disconnect' was between the picture and its corresponding numeric representation.

see Bransfield's writings.

Neurobiol Aging. 2005 May 12; [Epub ahead of print]

Beta-amyloid deposition and Alzheimer's type changes
induced by Borrelia spirochetes.

Miklossy J, Kis A, Radenovic A, Miller L, Forro L, Martins R, Reiss K, Darbinian N, Darekar P, Mihaly L, Khalili K.

Kinsmen Laboratory of Neurological Research, University of British Columbia, 2255 Wesbrook Mall, Vancouver, BC, Canada V6T 1Z3; University Institute of Pathology, Division of Neuropathology, University Medical School (CHUV), 1011 Lausanne, Switzerland.

The pathological hallmarks of Alzheimer's disease (AD) consist of beta-amyloid plaques and neurofibrillary tangles in affected brain areas.

The processes, which drive this host reaction are unknown. To determine whether an analogous host reaction to that occurring in AD could be induced by infectious agents, we exposed mammalian glial and neuronal cells in vitro to Borrelia burgdorferi spirochetes and to the inflammatory bacterial lipopolysaccharide (LPS).

Morphological changes analogous to the amyloid deposits of AD brain were observed following 2-8 weeks of exposure to the spirochetes.

Increased levels of beta-amyloid presursor protein (AbetaPP) and hyperphosphorylated tau were also detected by Western blots of extracts of cultured cells that had been treated with spirochetes or LPS.

These observations indicate that, by exposure to bacteria or to their toxic products, host responses similar in nature to those observed in AD may be induced.