cyanobacteria (blue-green algae), has been linked to
neurodegenerative disease in indigenous communities in Australia and the South Pacific (Guam) and more recently to sporadic Amyotrophic Lateral Sclerosis (sALS) and
Alzheimer’s disease (AD) in Canada and the USA.
Cyanobacteria are ubiquitously distributed in terrestrial, fresh water and marine environments and
due, in some cases, to mismanagement of these resources
human exposure to BMAA is increasing.
Of great concern is recent data showing BMAA is
***bioconcentrated by aquatic species***
raising the possibility that we could be subject to ‘silent’ exposure to BMAA
in the absence of any readily identifiable cyanobacterial blooms.
“It has been suggested that ***bb0666 ***encodes a putative N-acetylmuramyl-L-***alanine*** amidase involved in peptidogylcan synthesis critical for cell division.”
Why does that number not surprise me? Does the berberine metabolite mess with Bb's 16s (r)RNA?
Ribosomal *(r)* RNA genes are likely to be critical for survival and persistence in its hosts.
It appears berberine is metabolized to berberrubine which interacts with HP14, a
substrate of RNA
involved in RNA folding. (Linked previously).
P.S. Viruses such as HIV have RNA genomes that can be converted into DNA by an enzyme by an enzyme called reverse transcriptase.
Posted by Razzle (Member # 30398) on :
So this means that the RNA that can be converted into DNA may ultimately become incorporated into a cell as part if it's DNA? Is this how Viruses "hijack" cells to reproduce?