luvs2ride
Frequent Contributor (1K+ posts)
Member # 8090
posted
bummer!
-------------------- When the Power of Love overcomes the Love of Power, there will be Peace. Posts: 3038 | From america | Registered: Oct 2005
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posted
I wish that they would have given some details about how long he has had lyme, and what kind of therapy he has had to go through over the years so that this might have opened up the eyes of the general public to how serious this disease is! I'm sure that most people will think that ALS was the killer. Little do they know that it's all part of the same deal. Another death from lyme swept under the rug so to speak. My 2c's
-------------------- You're only a failure when you stop trying. Posts: 945 | From U.S | Registered: Oct 2004
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quote:Originally posted by micul: Little do they know that it's all part of the same deal. Another death from lyme swept under the rug so to speak. My 2c's
ditto, the more we look the more we find borrelia behind diseases such as ALS,Alzheimers,Fibro,etc.
Posts: 731 | From Humble,TX | Registered: Feb 2005
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Mathias
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Member # 5298
posted
I wish they would worded it: died of ALS symptoms of lyme disease or lyme disease with an ALS presentation. I don't know how many people need to die before they figure out there is a link between the two. There is no way he had both LD and ALS.
-------------------- Mathias Posts: 1250 | From New Jersey | Registered: Feb 2004
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quote:Originally posted by Mathias: I wish they would worded it: died of ALS symptoms of lyme disease or lyme disease with an ALS presentation.
If it's a "genetic" disease I'd call it a borrelia infection causing ALS symptoms. "Lyme" implies a zoonotic disease which may or may not be the case.
Posts: 731 | From Humble,TX | Registered: Feb 2005
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posted
ASL is short for discription of symptoms, not a dx of a disease. Lyme is a disease. Too bad doctors don't know and treat accordingly. Most likely, and this is just a guess, he died of fibgrin(sp) or a thicking of the blood which then traveled to his lungs . Paper said respertiory failure. Some one has posted from time to time the study and research and paper written by a Dr. in Mo. that explains this process nicely. TAKE BLOOD THINNER if you have this condition.
Posts: 219 | From Aubur,Al. USA | Registered: Oct 2004
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posted
ASL is short for discription of symptoms, not a dx of a disease. Lyme is a disease. Too bad doctors don't know and treat accordingly. Most likely, and this is just a guess, he died of fibgrin(sp) or a thicking of the blood which then traveled to his lungs . Paper said respertiory failure. Some one has posted from time to time the study and research and paper written by a Dr. in Mo. that explains this process nicely. TAKE BLOOD THINNER if you have this condition.
Posts: 219 | From Aubur,Al. USA | Registered: Oct 2004
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posted
in one study, approx. 50% of the men diagnosed with als, were positive for borrelia. the study is mentioned here.http://actionlyme.org/ALS_&_Lyme_47%25.htm when at this site, you'll have to wait for the scanned in text book mention of this study to load.
a "hair-splitting analysis": the correlation is there,but "officially", its borrelial cause is not formerly attributed to als. technically, since "more often than not", a tick bite, or insect bite(s) of certain kinds result in multiple infections,then, in the absolute sense of the word, borrelia can not be called "THE" cause. Personally, i beleive otherwise, and that multiple infections from a tick/insect bite CAN BE/IS the cause of als. my tentative conclusion.
imo, the results of at least one study by atkinson-barr confirms this, as does too many of us who were diagnosed with als,and/or other tbds who, subsequent to the als diagnosis, tested positive for one ,or more tbds, and who recovered substantially after treatment.
for a cp of the atkinson-barr study of als and lyme, search this site, and its archives.
quote:Originally posted by Troup Brazelton: Most likely, and this is just a guess, he died of fibgrin(sp) or a thicking of the blood which then traveled to his lungs . Paper said respertiory failure. Some one has posted from time to time the study and research and paper written by a Dr. in Mo. that explains this process nicely. TAKE BLOOD THINNER if you have this condition. [/QB]
You mean this one??
"Hypercoagulation, or thrombophilia, may be defined as a greater tendency than normal for blood to coagulate, or clot. Of approximately 500 borreliosis patients that I have tested, 90 percent have hypercoagulation. Comparatively, only five percent of the general healthy population has hypercoagulation.
Two major aspects of hypercoagulation are infections and hereditary or genetic abnormalities. Chronic infections such as borreliosis, Epstein-Barr virus, cytomegalovirus, human herpes virus 6 and mycoplasma have all been associated with hypercoagulation. These infections appear to be the driving force behind the greater tendency to form blood clots. Infections may elevate levels of fibrinogen, fibrin, thrombin/antithrombin complexes, fragment 1+2, and Factor II (prothrombin) activity.
When you get a cut, the fibrinogen in your blood converts into fibrin, which in turn forms a mesh to create a blood clot to help stop the bleeding. Antithrombin binds to thrombin to form a complex to prevent thrombin from causing blood to clot. Antithrombin protects against too much clotting, when it binds thrombin.
When thrombin is forming a clot it produces fragment 1+2. Prothrombin (Factor II) converts into thrombin, which causes blood to clot. When these coagulation components are high, it indicates that the blood has a greater tendency to clot.
Inherited or genetic disorders may also predispose to blood clotting. Some of these hereditary defects include antithrombin activity deficiency, protein C activity deficiency, protein S activity deficiency, Factor II gene mutation, APC resistance (Activated Protein C resistance is also called Factor V Leiden deficiency), elevated lipoprotein(a) or Lp(a), elevated PAI-1 (Plasminogen Activator Inhibiter-1), and elevated homocysteine. Hereditary hypercoagulation test panels are abnormal in 74 percent of borreliosis patients, as opposed to about two to five percent of the general healthy population.
Treatment for hypercoagulation caused by infections is heparin, which is a blood thinner. Typically, heparin is given subcutaneously (under the skin) by injection twice a day in low doses for not more than nine months. It can also be compounded into a troche that dissolves in the mouth, but that is usually more expensive and is often less effective than injections.
Symptoms that improve with heparin are pain, fatigue, cognitive problems and neurological problems. About 80 percent of borreliosis patients feel better with heparin, and it has been a safe treatment so far. (One patient did develop bleeding from the rectum, but then a colonoscopy revealed a colon cancer that had not yet spread to the local lymph nodes. In other words, the heparin unmasked a hidden malignancy, so in this case the side effect was a blessing.)
Heparin is not only a blood thinner, it is also anti-inflammatory, antiviral, antibacterial, and may even be anti-cancer (unproven). Therapy with heparin usually lowers the level of the coagulation components fibrinogen, fibrin, thrombin/antithrombin complexes, fragment 1+2 and Factor II activity. This is desirable, because elevated levels of these coagulation components can cause decreased capillary blood flow, if they are high enough. Capillaries are microscopic blood vessels that are about eight microns wide. A normal red blood cell, which travels through the capillaries, is about seven microns wide. When elevation of coagulation components occur, they could conceivably attach to the inside surface (endothelial surface) of capillaries, thereby narrowing them. For example, fibrinogen attached to the inside surface could make it harder for a seven-micron-wide red blood cell to squeeze through the narrowed capillary. Reduced blood flow in capillaries would in turn reduce oxygen and nutrients, and reduce removal of toxins from tissues. It stands to reason that if heparin could improve blood flow, antibiotics and hormones would be more effective because they could pass through capillaries easier.
Life is in the blood. Less blood flow means less ``life,'' and possibly more symptoms and diseases -- perhaps even death.
Hypercoagulation is associated with other chronic diseases, not just borreliosis. It is my opinion that how hypercoagulation is treated will become a paradigm shift in medicine, once further research has been accomplished."
by Dr C of MO
Ordinary blood thinners won't work for this...has to be heparin [which works on the fibrinogen]...or lumbrokinase.
I used to take heparin, but opted for a more natural route after several years of that.
I tried taking nattokinase and Rutozyme, but my fibrinogen levels were still sky high. So now I'm taking lumbrokinase. I'll get tested again in 3 months.
-------------------- --Lymetutu-- Opinions, not medical advice! Posts: 96239 | From Texas | Registered: Feb 2001
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quote:Originally posted by psano: Is fibrinogen level the only blood test needed to check on this?
Hmmmm, that may be all you need. I was recently at Dr C's and he said he was no longer running the ISAC test. [I already had that run several years ago.]
He ran a test which checked only the fibrinogen level. Mine was "higher than high." ugh!
Troup...I'll have to send that to you in a PM.
-------------------- --Lymetutu-- Opinions, not medical advice! Posts: 96239 | From Texas | Registered: Feb 2001
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