GiGi
Frequent Contributor (5K+ posts)
Member # 259
posted
It was always known that bacteria and fungi could rapidly exchange DNA bits that are important for the development of drug resistance and other mechanisms important for their respective survival.
What has recently emerged is that viruses can also plug into this microorganism information network and borrow RNA and DNA strand-sections from bacteria that have learned tricks of how to invade the host's immune system, creating drug and immune system resistant monsters.
These types of viruses have been coined "viteria" - a combination of virus ("vi") and bacteria.
Antibiotics will be completely ineffective. The correct antiviral regime will successfully treat the condition.
(from: Relationship Between Chronic Illness and Microorganisms - discussed in more detail at a Dr. K. conference a few days ago)
Take care.
Posts: 9834 | From Washington State | Registered: Oct 2000
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cantgiveupyet
Frequent Contributor (1K+ posts)
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thanks Gigi, for this info.
-------------------- "Say it straight simple and with a smile."
"Thus the task is, not so much to see what no one has seen yet, But to think what nobody has thought yet, About what everybody sees."
-Schopenhauer
pos babs, bart, igenex WB igm/igg Posts: 3156 | From Lyme limbo | Registered: Oct 2005
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I've been doing a lot of reading about this recently -- I'm glad you brought it up.
I have had a hunch for a while that there must be a viral component (perhaps beyond the usual suspects of HHV, CMV, EBV) that is adding to the recalcitrance to abx treatment for so many of us.
Dr. John Martin has done a lot of work in this area -- but his findings have yet to be replicated, or taken seriously. I don't have enough bioscience under my belt to be a truly informed consumer of his research (I'm a social scientist) -- but to my layman's eye, it seems pretty sound.
I asked my LLNP to add an antiviral to my drug regimen, based just on my hunch, and she agreed to try it. After a less than 2 weeks on Valtrex, my fatigue levels have decreased to almost no fatigue -- amazing progress. I'm still battling the usual lyme stuff,(worse than usual at the moment) but at least the fatigue has diminished, which is huge.
I'm doing a ton of reading about virii and chronic illness at the moment.
-------------------- "Looks like freedom but it feels like death.. It's something in between, I guess"
Leonard Cohen, from the song "Closing Time" Posts: 822 | From California | Registered: Jan 2006
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treepatrol
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Member # 4117
The term viteria has been introduced to reflect the dual contribution of genes from viruses infectious for eukaryotic cells and genes from bacteria and/or bacterial plasmids. Viteria are presumably infectious for both prokaryotic and eukaryotic cells. The diverse range of metabolic processes presumably could provide a selective growth advantages to an infected bacteria. In a preliminary study, cytopathic transmissible agents were recoverable from an extract of fecal bacteria obtained from the patient from whom the prototype stealth virus was cultured. The ability of this agent to alter the metabolic profile of normal bacteria is being assessed. Similarly, human cells infected with the agent will be tested for the expression of bacterial related sequences.
Positive preliminary findings for such agents have also been obtained in fecal extracts from several stealth virus infected patients and also in the feces of symptomatic animals. As noted above, the assimilated bacterial genes may provide selective growth advantages to an infected bacteria. Efforts are, therefore, underway to selectively propagate viteria infected bacteria using nutritionally deprived media. In related studies, the potential of fungal organisms as an additional permissive host for viteria is being explored. One of the genes identified in the prototype stealth virus does, in fact, show good homology to a gene from the fungal organism, Arabidopsis thaliana. If fungal derived genes were to be predominant and/or if the viruses were to show a propensity to replicate in fungi, the term vifungus might be preferable to viteria.
The potential of viteria being harbored within the bacteria and/or fungal flora of the body has obvious diagnostic, therapeutic and epidemiological implications. Diagnostically, the presence of bacterial sequences in viteria can help reconcile the inconsistent reports of positive mycoplasma and chlamydia PCR assays in patients with CFS and, with the related Gulf war syndrome. Similarly, the laboratory findings suggesting that patients labeled as having chronic Lyme disease are infected with Borrelia, could be explained by viteria expressing amino acids and/or nucleotide sequences cross reactive with those of Borrelia. Indeed, the majority of chronic Lyme disease patients so far tested have yielded a positive result in assays for stealth viruses.
Therapeutically, it is useful to determine the antibiotic susceptibility of any bacteria cultured from the bowel, mouth or other locations that can be shown to be viteria infected. This should help control possible relapses in a patient treated with anti-viral agents, and more importantly, help control the spread of infection within households and within larger community groups. Antibiotic suppression of metabolically aberrant, viteria-infected bacteria, may also reconcile reports of clinical improvements occasionally seen in patients diagnosed as having CFS, Gulf war syndrome, Lyme disease, etc. To rationalize such therapies, it will be useful, however, to determine the specific antibiotic susceptibilities of viteria infected bacteria and/or fungi isolated from a patient. Analysis of viteria load can also help direct various dietary and other interventions that can help influence the bacterial composition of the bowel and other body sites.
Potentially Oncogenic Viteria
Stealth viruses have the capacity to assimilate various cellular genes, including genes with potential oncogenic activities. For example, multiple copies of a melanoma associated oncogene were detectable in the prototype SCMV-derived viteria. Positive stealth virus cultures have now been obtained in patients with various malignancies, including multiple myeloma, lymphomas, gliomas, breast cancers, lung cancers, melanomas and salivary gland tumors. Many of the patients describe disabling neurocognitive and mood disorders in themselves prior to the diagnosis of malignancy, and importantly, in some of their family members. As an example, a mother who struggles with a chronic fatigue syndrome, and whose son developed an acute and persisting learning disorder as a Junior in high school, reported that after years of being depressed, her father had been diagnosed with multiple myeloma. The mother subsequently developed a uterine malignancy and was incidentally found to give a false positive antibody test for HIV. All of family members are positive by stealth virus culture. Sequencing studies on the virus isolates from these patients may help establish a common source with subsequent differences in assimilated additional genes. Sequencing studies also need to be performed on markedly cytopathic agents cultured from malignant breast tissue, bone marrow, lymph nodes, brain biopsies, etc., of various cancer patients tested over the last several years. It is predicted that such studies will reveal additional examples of stealth adapted viruses containing cellular genes with oncogenic activity. The possibility that such viruses might also be carried by bacteria has ominous public health implications. It may, however, hasten the testing of various therapeutic strategies with a dual focus on their effects on the cancer and on any underlying neurological/neuropsychiatric disorders. An awareness of potentially oncogenic viteria may also help curtail the spread and possible further introduction of viteria into humans and animals.
The Origins of Viteria
A critical question is whether viteria are products of the late 20th century, or have been in existence for eons and have simply previously gone unrecognized. As documented above, the alignments of various bacterial genes within several of the clones of the prototype stealth virus is quite different than that so far encountered in known bacteria. This points to a more recent origin of the stealth virus. Overall, the available sequencing data are indicative of a complex progressive process of genomic recombinations between portions of the original SCMV genome and genes of diverse bacterial species. If this conclusion is substantiated with additional isolate, extremely urgent consideration must be given to the potential capacity of viteria to drastically reformat the genomic structures of both prokaryotic and eukaryotic organisms.
As discussed elsewhere, the SCMV sequences in the prototype stealth virus point to an African green monkey as the primary source of this particular isolate. These monkeys were widely used, and indeed are still used to produce an attenuated live polio virus vaccine. Detailed information on the process of vaccine production and on the results of Government safety testing of vaccines is shielded from public inquiry as a protection of proprietary interests. For inexplicable reasons, molecular assays for SCMV contamination of production lots of polio vaccines have not been employed. Unquestionably, an open discussion of the present and prior risks inherent in the production and use of polio vaccines, and of other live vaccines administered to human and animal, could shed useful insights into a possible calamity of modern vaccine technology.
Animals were presumably also used in the production and testing of biological agents for military purposes. As detailed in a recently published book, Brucella bacteria have been considered as potential pathogens for germ warfare. This observation is intriguing since nucleotide and amino acid sequences from this bacteria are present within the cloned genes derived from the viteria infected patient. Records for biological studies performed with these and other bacteria and viruses should also be made available for scientific review.
Conclusion
The existence of viteria extends the unsettling notion that stealth viruses represent "Nature's Biological Weapons Program." Continued, unchecked replication of genetically diversified, potentially oncogenic, pathogens, that can pass between species, yet bypass cellular immune defenses, could have devastating consequences. A program to combat viteria through a better understanding of their origins and biological activities is urgently required. Further information on stealth-adapted viruses, and on the viteria sequencing program, is available at www.ccid.org
-------------------- Do unto others as you would have them do unto you. Remember Iam not a Doctor Just someone struggling like you with Tick Borne Diseases.
-------------------- When the Power of Love overcomes the Love of Power, there will be Peace. Posts: 3038 | From america | Registered: Oct 2005
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stymielymie
Frequent Contributor (1K+ posts)
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posted
i have been telling you guys for at least 2 years that anti-virals were necessary.
they serve a dual purpose, one to prevent superinfection of herpes, ebv, hpv, and flu viruses. it can also prevent getting Hodgkin's lymphoma, shingles.
the antivirals also prevent the bacteria from changing and exchanging dan,rna fragments to other cells. this helps prevent viteria, bacteria mutation to viruses, and our cells picking up rna fragments from bb and hiding in the tissues, and from white blood cells and macrophages.
i started using them 2 years ago when i found that bacteria could mutate to viruses. in my research the bb count possibly mutate and hide in deep tissues,very simmilar to herpes viruses.
herpes will stay in the nerve ending and expose itself and become potent when the body is under stress, mental, physical,emotional
my lyme kick its head, after being pretty much dormant for 7 years. it hit me when my dad past away and still kicking me. when your down it kicks more and more.
so thats my story and ideas.
docdave docdave.
Posts: 1820 | From Boone and Southport, NC | Registered: Sep 2006
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GiGi
Frequent Contributor (5K+ posts)
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posted
Minimoney, If I remember correctly, Dr. Martin after a three-day Klinghardt Conference dealing mainly with Lyme and Co. around 2000, said he wished he had never called it a "stealth". A lot is happening in this field almost daily.
Posts: 9834 | From Washington State | Registered: Oct 2000
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GiGi
Frequent Contributor (5K+ posts)
Member # 259
posted
Here is more from Dr. K. on the Relationship Between Chronic Illness and Microorganisms from our last conference a few days ago:
"When a microorganism is not recognized by the immune system, or not completely recognized, the guest will survive in the hoast. I believe believes that all of us are home to many species of pathogenic microorganisms that include large parasites such as tape-, hook-, and roundworm, fungi such as aspergillus and Candida, smaller mostly single-cell organisms such as giardia, amoebas, cryptosporidium, even smaller cell wall deficient bugs( which often were created by taking antibiotics too often) evil creatures such as mycioplasms and Chlamydia, and of course bacteria such as salmonella, Borrelia, several species of strep and staph, and finally nanobacteria which are found in the arterial plaque and then, last but not least: the viruses.
The most important viruses clinically are the 8 herpes viruses and the different strains of influenza In addition, there are the odd ones: Simian virus 40 (SV 40) was introduced into our bodies inadvertently with the polio vaccine.
The prions causing mad cow disease (BSE) are no longer a European problem only. A study on deceased Alzheimer patients in the US showed that 20% had prions on their brain; it is clear from this list that we need to focus on prevention, and that we need to find treatments that are rapid, non-invasive and effective.
(There is no other diagnostic method that can rival ART in speed, non-invasiveness and accuracy.)
Common connections (microorganisms listed in order of how often they are found):
ALS: Mycoplasma, Borrelia, HSV-1 and 2, CMV, HHV-6, retroviruses
Coronary heart disease and stroke: Herpes viruses, Chlamydia, H. pylori, strep and other germs from the oral cavity (over 300 known microorganisms in a normal mouth), Nanobacteria, Salmonella
Diabetes type I: influenza, CMV, childhood vaccines, Borna and Rotaviruses
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