posted
A new study suggests that green tea extracts make abx more effective. Anyone have any experiences with this combo?
"In collaboration with professors at Montclair State, professor Chu has shown that when combined with antibiotics in vitro, thus modified green tea extracts can boost the effectiveness of some of the most commonly prescribed antibiotics such as Ampicillin by up to more than 400% when compared to antibiotics alone. Somewhat characteristic was the impact that a modified green tea extract had with Bacitracin, Chloramphenicol and Rifampin on S. marcescens, scoring a 292%, 119%, and 225% respective increase in inhibition (effectiveness)."
It seems to contradict this earlier study at least in mice fighting MRSA with amoxicillin:
"This study concluded that tea extract weakened the antibacterial effect of amoxicillin in MRSA infected mice. Tea drinking is not recommended in combination with amoxicillin treatment."
poppy
Frequent Contributor (1K+ posts)
Member # 5355
posted
So how are patients/doctors supposed to figure these things out if we are faced with contradictory evidence? Or does this mean some abx work better and some don't? Gack.
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posted
Geez. the way of the world these days. One thing is good for you one day, bad the next.
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Keebler
Honored Contributor (25K+ posts)
Member # 12673
posted
- Just my thoughts: well, there is nothing that could ever stop me from enjoying several cups of organic green tea a day. For thousands of years, it has provided benefit.
Perhaps, were I to be taking Rx, I might not drink a cup with the Rx, but wait a little while. Telling people to not even drink tea, though, is like telling them not to eat colorful vegetables or bright deep colored berries that also have similar qualities of support to body organs.
I also think that green tea need not be messed with regarding making it an "extract". -
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Marnie
Frequent Contributor (5K+ posts)
Member # 773
posted
Watch the specific antibiotic - green tea extract synergy.
Ampicillin, NOT amoxicillin to fight MRSA.
Epigallocatechin gallate synergy with ***ampicillin***/sulbactam against 28 clinical isolates of methicillin-resistant Staphylococcus aureus
epigallocatechin gallate (EGCg, a main constituent of tea catechins)
Serotonin has a blocking effect on sodium channels *in human airway epithelial cells*, but the receptor subtype involved has not been established (Graham et al., 1992).
Alpha-1 antitrypsin, which protects the body from a powerful enzyme called neutrophil elastase.
Neutrophil elastase is released from white blood cells to fight infection,
but
it can attack normal tissues (especially the lungs)
if not tightly controlled by alpha-1 antitrypsin.
***EGCG enhances the expression of alpha-1 antitrypsin***
by regulating the PI3/AKT pathway.
(EGCG = epigallocatechin-3-gallate)
Persons can have genetically low levels of this enzyme and genetic testing for (low levels of) alpha-1 antitrypsin is FREE (at least in my pulmonologist's office). It involved a simple skin pin pick and the blood sample was sent out to a lab.
I just did it - I'm reacting big time to red tide. Coricidin HBP for coughs and colds helps counter the toxins in red tide that impact the sodium channels (open). It is a H1 blocker too without the "sleepy" aspect.
The toxins released by red tide open the sodium channels and trigger histamine release.
(The toxins T17 and T34 are the ones impacting the sodium channels.)
This ***brevetoxin-induced bronchospasm*** was effectively blocked by
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