Many patients are told that they have Multiple Sclerosis (MS) because of brain MRI findings or a spinal tap was positive for oligoclonal bands (OCB) or myelin basic protein (MBP). The medical literature is quite emphatic that MRI does not reliably distinguish between MS an LD because there is too much overlap in their supposedly distinct appearance and location of plaques. Plaques have been detected with both disorders in the brain and spinal cord. OCB's and MBP are non-specific markers for demyelination (loss of sheath around nerves) and do not signify a cause of the demyelination. In Miklossy's study above, senile plaques stained avidly for Bb spirochetes. Vincent Marshall reviewed the MD literature in Medical Hypothesis (Vol 25: 89-92, 1988) and advances the notion that LD is causing MS! His survey revealed that multiple studies prior to 1951 were able to demonstrate spirochetes in the spinal fluid of MS patients (by inoculation into animals and on silver stain of CNS tissues). Dr. Coyle has documented the presence of antibodies to Bb in MS patients (Neurology Vol. 39:760-763, 1989). The encephalopathy attributed to MS is very reminiscent of LD. Both MS and LD are associated with sinusitis (Lancet, 1986). Dr. Leigner has reported a case of LD which fulfilled all criteria for MS. The epidemiology of MS and the geographic distribution parallels that of LD. The symptoms of both LD and MS can be aggravated if the patient takes a hot bath. Anecdotally, patients with LD, who previously had been identified as MS, responded to antibiotic therapy.
Half way down artical.
http://cassia.org/essay.htm

Encephalitis
Definition

Encephalitis is an inflammation of the brain, usually caused by a direct viral infection or a hypersensitivity reaction to a virus or foreign protein. Brain inflammation caused by a bacterial infection is sometimes called cerebritis. When both the brain and spinal cord are involved, the disorder is called encephalomyelitis. An inflammation of the brain's covering, or meninges, is called meningitis.

Description

Encephalitis is an inflammation of the brain. The inflammation is a reaction of the body's immune system to infection or invasion. During the inflammation, the brain's tissues become swollen. The combination of the infection and the immune reaction to it can cause headache and a fever, as well as more severe symptoms in some cases.

Approximately 2000 cases of encephalitis are reported to the Centers for Disease Control in Atlanta, GA each year. The viruses causing primary encephalitis can be epidemic or sporadic. The polio virus is an epidemic cause. Arthropod-borne viral encephalitis is responsible for most epidemic viral encephalitis. The viruses live in animal hosts and mosquitos that transmit the disease. The most common form of non-epidemic or sporadic encephalitis is caused by the herpes simplex virus, type 1 (HSV-1) and has a high rate of death. Mumps is another example of a sporadic cause.

Causes & symptoms
Causes

There are more than a dozen viruses that can cause encephalitis, spread by either human-to human contact or by animal bites. Encephalitis may occur with several common viral infections of childhood. Viruses and viral diseases that may cause encephalitis include:

Chickenpox
Measles
Mumps
Epstein-Barr virus (EBV)
Cytomegalovirus infection
HIV
Herpes simplex
Herpes zoster (shingles)
Herpes B
Polio
Rabies
Mosquito-borne viruses (arboviruses).

Primary encephalitis is caused by direct infection by the virus, while secondary encephalitis is due to a post-infectious immune reaction to viral infection elsewhere in the body. Secondary encephalitis may occur with measles, chickenpox, mumps, rubella, and EBV. In secondary encephalitis, symptoms usually begin 5-10 days after the onset of the disease itself and are related to the breakdown of the myelin sheath that covers nerve fibers.

In rare cases, encephalitis may follow vaccination against some of the viral diseases listed above. Creutzfeldt-Jakob disease, a very rare brain disorder caused by an infectious particle called a prion, may also cause encephalitis.

Mosquitoes spread viruses responsible for equine encephalitis (eastern and western types), St. Louis encephalitis, California encephalitis, and Japanese encephalitis. Lyme disease, spread by ticks, can cause encephalitis, as can Colorado tick fever. Rabies is most often spread by animal bites from dogs, cats, mice, raccoons, squirrels, and bats and may cause encephalitis.
http://www.chclibrary.org/micromed/00046650.html

Thats why there finding myelinbreakdown of the myelin sheath that covers nerve fibers.

Another
Multiple sclerosis (MS) is a disease of the central nervous system (CNS), the nerves that comprise the brain and spinal cord. It has two major features:

The primary characteristic of this disease is the destruction of myelin, a fatty insulation covering the nerve fibers. The end results of this process, called demyelination, are multiple patches of hard, scarred tissue called plaques. (Multiple sclerosis is well named. Sclerosis comes from the Greek word skleros, which means hard.)

Myelin is the layer that forms around nerves. Its purpose is to speed the transmission of impulses along nerve cells.

Another important feature in the disease is destruction of axons -- the long filaments that carry electric impulses away from a nerve cell -- which is now considered to be a major factor in the permanent disability that occurs with MS.
http://www.enh.org/healthandwellness/encyclopedia/wellconnected/000017.asp

READ why. http://www.canlyme.com/lymemultiplesclerosis.html

Worldwide, MS prevalence parallels the distribution of the Lyme disease
pathogen Borrelia (B.) burgdorferi, and in America and Europe, the
birth excesses of those individuals who later in life develop MS
exactly mirror the seasonal distributions of Borrelia transmitting
Ixodes ticks. In addition to known acute infections, no other disease
exhibits equally marked epidemiological clusters by season and
locality, nurturing the hope that prevention might ultimately be
attainable.

As minocycline, tinidazole and hydroxychloroquine are reportedly
capable of destroying both the spirochaetal and cystic L-form of B.
burgdorferi found in MS brains, there emerges also new hope for those
already afflicted. The immunomodulating anti-inflammatory potential of
minocycline and hydroxychloroquine may furthermore reduce the Jarisch
Herxheimer reaction triggered by decaying Borrelia at treatment
initiation.

Even in those cases unrelated to B. burgdorferi, minocycline is known
for its beneficial effect on several factors considered to be
detrimental in MS. Patients receiving a combination of these
pharmaceuticals are thus expected to be cured or to have a longer
period of remission compared to untreated controls.

Although the goal of this rational, cost-effective and potentially
curative treatment seems simple enough, the importance of a
scientifically sound approach cannot be overemphasised.
http://www.talkaboutsupport.com/group/alt.support.mult-sclerosis/messages/365272.html