dontlikeliver
Frequent Contributor (1K+ posts)
Member # 4749
posted
How come, assuming that the same animals from which BB comes from (through the ticks), we don't also get something like Rabies (from Racoons, squirrels and the like)??
Posts: 2824 | From The Back of Beyond | Registered: Oct 2003
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Linda LD
Frequent Contributor (1K+ posts)
Member # 6663
posted
?
Posts: 1171 | From Knoxville, TN US | Registered: Dec 2004
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treepatrol
Honored Contributor (10K+ posts)
Member # 4117
posted
I read years ago sunlight ultiviolet light kills the virus.But some trappers checking traps in the dark have contracted rabies from the mouth of the den hole the steam in winter coming out in the dark can carry live virus.
Also while researching your rabies question I found something that people using this should be aware of. Quote Chloroquine, a prophylactic treatment for malaria, is apparently iatrogenic for rabies. In other words, prophylactic treatment with chloroquine for malaria, while protecting against malaria, reduces the body's ability to create antibodies for rabies and other diseases. For example, three months after vaccination against rabies, chloroquine users have approximately half the antibody response to rabies that non-chloroquine users have. Patients who have been vaccinated against rabies can die from rabies if they are also chloroquine users.FROM: http://www.tarakharper.com/v_rabies.htm#des
Thats very interesting lowered immune responce hmmm. Also found this.
Qoute
New search-and-destroy virus strikes HIV cells; Modified rabies strain kills only those infected with AIDS
The San Francisco Examiner - Friday, Sept. 5, 1997 Lisa M. Krieger, Examiner Medical Writer
-------------------------------------------------------------------------------- Fighting fire with fire, scientists are enlisting the help of other infectious viruses to seek and destroy cells harboring the AIDS virus. A team from Yale University Medical School announced in Friday's issue of the journal Cell that they have built a strain of virus with the uncanny ability to kill cells infected with HIV, but not healthy bystander cells.
The engineered virus reduced HIV to undetectable levels, raising the possibility of an entirely new kind of therapy for treating AIDS and maybe other viral diseases.
Although the work was done in the lab and is not yet ready to be tried in human patients, "the reports are highly significant as they represent a leap forward in how we might apply basic understandings of viral biology to the design of therapies," wrote Stanford University Medical Center's Dr. Garry P. Nolan in an accompanying editorial.
The creation of anti-virus viruses "boldly demonstrates that it is now possible to reverse the aim of the biochemical grappling hooks that HIV uses to enter cells - and to employ them against the virus itself," according to Nolan.
The approach is exciting because it employs the exact chemical cues that HIV uses, making it difficult for the AIDS virus to sidestep the assault.
Moreover, the highly selective therapy - engineered to go only where it does good, not bad, thus reducing nasty side effects - holds potential for treatment of other diseases, not just AIDS, according to scientists.
"It's like a conversion from traditional bombing to laser-guided smart bombs," Dr. Ronald Desrosiers of the Harvard Medical School told the New York Times. "This virus doesn't seem to infect any cells other than those infected with HIV."
Ironically, the hunter-seeker virus is derived from domesticated versions of an ancient enemy of humans: the rabies virus. This rabies relative, called vesicular stomatitis virus (VSV), is a cattle virus and not lethal to humans.
Scientists first disabled the VSV so it couldn't infect its usual cellular targets.
Then they inserted into the virus some genes for HIV-specific receptors, which are proteins on the surface of immune system cells that permit entry and infection by the AIDS virus.
Working from this new set of instructions, the chimeric virus then sought out, entered and killed human cells that showed signs of hidden HIV infection.
"This is a classic case of predator-prey population dynamics at the molecular level," according to Nolan.
Remarkably, the engineered virus even replicates in cells - becoming, in a sense, a live therapy that is capable of multiplying and proliferating, not only killing bad cells but then moving on to kill others.
The VSV, created by Dr. John K. Rose of Yale, ultimately cut HIV production more than 300-fold to levels that were barely detectable or undetectable.
A similar approach using a modified rabies virus to attack HIV-infected cells has been developed at the Federal Research Center for Virus Diseases of Animals in Tuebingen, Germany, and is also reported in Friday's Cell. But this rabies virus does not kill the infected cells.
At this early point in the research, it is premature to introduce the new viruses into humans, even if intended for good purposes, noted Nolan.
This is because the new viruses might affect the health of an immunocompromised patient. They might spread to unintended cells or be transferred, accidentally, to ther people.
The regulatory authorities at the federal Food and Drug Administration, among others, are likely to raise their eyebrows at the prospect of viral therapy.
The next hurdle is for the therapy to be tried in animals, in this case monkeys infected with the monkey version of HIV, known as simian immunodeficiency virus or SIV. From: http://www.aegis.com/news/SFE/1997/SE970902.html
-------------------- Do unto others as you would have them do unto you. Remember Iam not a Doctor Just someone struggling like you with Tick Borne Diseases.
posted
Maybe because rabies is strictly a warm-blooded animal thing? And ticks aren't warm-blooded critters, so maybe that inactivates it?
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