posted
Friends, I was just thinking to remind you that LD can cause an ACUTE TRANSVERSE MYELITIS. This is how my situation was uncovered. You may want to visit myelitis.org and have a look see at the situation. This is very important for all of us to know. It seems that ATM is the usual route for this problem turning neurological.
this is one topic i don't recall being addressed, if at all.
http://www.mucos.de did a study on myelitis, utilizing one, or more kinds of multi-enzyme formulations for this, with at least very good to excellent results.
-------------------- We have only this moment, sparkling like a star in our hand... and melting like a snowflake. Let us use it before it is too late. Posts: 221 | From the hills | Registered: Mar 2006
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posted
In the hopkins article at the website www.myelitis.org they exclude infectious causes from the group they are studying as TM, and call those infectious myelitis instead. I wonder what the differences in presentation would be? Guess I will have to read the whole long article some time looking for the answer.
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treepatrol
Honored Contributor (10K+ posts)
Member # 4117
1. Development of sensory, motor or autonomic dysfunction attributable to the spinal cord 2. Bilateral signs and/or symptoms 3. Clearly-defined sensory level 4. Inflammation within the spinal cord demonstrated by CSF pleocytosis or elevated IgG index or gadolinium enhancement {If none of the inflammatory criteria is met at symptom onset, repeat MRI and LP evaluation between 2-7 days following symptom onset}
Exclusion criteria 1. History of previous radiation to the spine within the past 10 years 2. Clear arterial distribution clinical deficit consistent with thrombosis of the anterior spinal artery 3. Extra-axial compressive etiology by neuroimaging {MRI of spine preferred. CT myelography acceptable. X-ray, CT of spine are not adequate} 4. Abnormal flow voids on the surface of the spinal cord c/w AVM 5. Serologic or clinical evidence of connective tissue disease {sarcoidosis, Behcet's disease, Sjogren's syndrome, SLE, mixed connective tissue disorder etc} {Diagnostic of Connective-Tissue Associated TM} 6. History of clinically apparent optic neuritis {Diagnostic of Neuromyelitis optica} 7. CNS manifestations of syphilis, Lyme disease, HIV, HTLV-1, mycoplasma, other viral infection {e.g. HSV-1, HSV- 2, VZV, EBV, CMV, HHV-6, enteroviruses} {Diagnostic of Infectious myelitis } 8. Progression to nadir in less than 4 hours from symptom onset 9. Symptom progression continues beyond 21 days from symptom onset 10. Brain and spinal cord MRI abnormalities suggestive of MS and presence of oligoclonal bands in CSF {Suggestive of TM associated with MS. Apply McDonald criteria to definitively define MS} AVM= arteriovenous malformation; SLE= systemic lupus erythematosus; HTLV-1= human T-cell lymphotropic virus-1; HSV= herpes simplex virus; VZV= varicella zoster virus; EBV=Epstein-Barr virus; CMV= cytomegalovirus; HHV= human herpes
Heres there excusion technics:
Detection of lyme disease of the CNS typically is based on antibody detection methods {ELISA with confirmatory western blot} and the CSF/serum index is often helpful in determining whether there is true neuroborreliosis
-------------------- Do unto others as you would have them do unto you. Remember Iam not a Doctor Just someone struggling like you with Tick Borne Diseases.
treepatrol
Honored Contributor (10K+ posts)
Member # 4117
posted
History of acute transverse myelitis Several cases of `acute myelitis' were described in 1882, and pathological analysis revealed that some were caused by vascular lesions and others by acute in�ammation {1,2}. In 1922 and 1923, physicians in England and Holland became aware of a rare complication of smallpox vaccination: inflammation of the spinal cord and brain {3}. Given the term `post-vaccinal encephalomyelitis', over 200 cases were reported in those 2 years alone. Pathological analyses of fatal cases revealed in�ammatory cells and demyelination. In 1928, it was first postulated that many cases of acute myelitis are `postinfectious rather than infectious in cause' because for many patients the `fever had fallen and the rash had begun to fade'{{ah ever present the rash interesting}}} when the myelitis symptoms began {4}. It was proposed, therefore, that the myelitis was an `allergic' response to a virus rather than the virus itself that caused the spinal cord damage. It was in 1948 that the term `acute transverse myelitis' was utilized in reporting a case of fulminant inflammatory myelopathy complicating pneumonia {5}. From 4th Artical:
-------------------- Do unto others as you would have them do unto you. Remember Iam not a Doctor Just someone struggling like you with Tick Borne Diseases.
posted
Meningo-encephalomyelitis (sp.?) is associated with Lyme Disease, right? It took over a year for me to figure that out by reading medical journal citations here on LymeNet, then found out it's written of in 'Lyme Disease--Everything you Wanted to Know...' by Karen Forschner.
So would that simply be chronic vs. acute?
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LostCityAgent
Unregistered
posted
Good morning, I have read over all of your posts. I can tell you that it is known/accepted in the widespread neurological community of physicians and researches that LD, syphilis, MS, et al. are commonly responsible for Acute Transverse Myelitis. I have read nothing at John's Hopkins but I can tell you that it is a fact that LD can be an underlying cause for ATM. My supposed attack with ATM was mild in comparison to those left in wheel chairs and on venelators.
Just one example referring to (chronic) meningoencephalomyelitis.
I would like to hear of those here who experienced acute phase. Recall one person writing that they lost use of their legs quickly over 3 days.
Suppose people could write all sorts of things, but it does seem believable.
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LostCityAgent
Unregistered
posted
Sparky, It seems that this presents itself in an imitation of ATM? I ask this because I had the onset symptoms of ATM but when I just had a t-spine MRI nothing showed up, potentially also to my body habitus.
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