Your recent posts have been invaluable regarding supplements needed to help fight lyme.
Because of the research you've found showing that borrelia bacteria feed on zinc, I know we'll have to make the choice of avoiding zinc, so as to not feed the BB, or supplementing zinc, because zinc is necessary for the immunce system to work.
This fits with the article you posted last week about Bb crippling the immune system -- if Bb takes our zinc & zinc is necessary to our immune system function, then Bb prevents the immune system from destroying it.
I am thinking I need to support the immune system with zinc so it can fight the Bb, even though the Bb will be stronger too. Is that a bad decision?
Also, any idea what type of zinc supplement would be best? Zinc sulfate?
Thank you so much for your research & explanations. Most of us could not understand it ourselves without you.
posted
I take 15-30mg of zinc/copper every day, and am able to stay in Lyme remission on maintenance abx. The zinc recharges not only the immune system, but also sex drive, so unless there is a dire warning from the CDC stating otherwise, the zinc stays put.
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Centrum Silver contains 100% of the RDA...the lowest amt. we need to survive. Recommended Dietary Allowance...this is daily allowance. There is, however, a difference for many nutrients if you compare the RDA with ODA...optimal dietary allowance.
To play it safe, go with that which is in "daily" formulas.
Pharmanex vitamin packets (a daily packet contains 5 pills)...contains 67% of the RDA for zinc.
So many nutrients are imbalanced!!! What a mess.
While trying to figure out how to raise bicarbonate levels...safely (!)...I also found the following this morning:
o The clinical presentation of thiamine deficiency is characterized by severe acidosis with markedly elevated lactic acid levels and by shock, which is often resistant to inotropic agents and volume resuscitation. Thiamine acts as a cocarboxylase, catalyzing decarboxylation of pyruvic acid and acetyl-coenzyme A (CoA).
It also acts as a coenzyme for pyruvate dehydrogenase activity and oxidative decarboxylation of alpha ketoglutarate to succinyl CoA.
Thiamine deficiency causes excessive pyruvate and impaired fatty acid metabolism through the Krebs cycle. Generation of nicotinamide adenine dinucleotide (NADH) in the Krebs cycle is also impaired, stimulating anaerobic glycolysis and leading to even more lactate production.
Thiamine deficiency produces polyneuropathy and weakness or paralysis, as well as cardiac failure known as cardiac beriberi. Lack of thiamine intake can lead to depleted stores within 10 days.
o Thiamine deficiency is the likely diagnosis for patients who have been on total parenteral nutrition for 2 weeks or longer and who develop metabolic acidosis, lactic acidosis, and shock resistance to inotropic support. Thiamine administration rapidly corrects the clinical symptomatology.
� THAM (tris-hydroxy-methyl-amino-methane) is a buffer that can be used to treat acidosis when concerns exist regarding
CO2 accumulation from the metabolism of administered sodium bicarbonate.
THAM increases serum bicarbonate predictably and rapidly, as follows: o THAM + H2CO3 � THAM-H + HCO3 o H2CO3 � CO2 + H2O
Alkalinizing agents -- Sodium bicarbonate is used as a gastric, systemic, and urinary alkalinizer and has been used in the treatment of acidosis resulting from metabolic and respiratory causes, including diabetic coma, diarrhea, kidney disturbances, and shock.
Alternatively, tris(hydroxymethyl)aminomethane (tromethamine, THAM) is a buffering agent that increases pH without increasing levels of PCO2. It may be used to correct metabolic acidosis if sodium bicarbonate is contraindicated.
B1 = thiamine.
Antibiotics, sulfa drugs and oral contraceptives may decrease thiamine levels in the body.
Posts: 9426 | From Sunshine State | Registered: Mar 2001
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