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» LymeNet Flash » Questions and Discussion » Medical Questions » MS and Borrelia

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Author Topic: MS and Borrelia
johnnyb
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Some Interesting articles I found. Anyone want to add anything?

http://www.shoptown.com/Dean/ALS2Lyme.htm

http://home.pon.net/caat/lyme/cyst_phase/MS_study.htm


search for in this page Borrelia mylophora:
http://www.diagnose-me.com/cond/C63897.html

- JB

Posts: 1197 | From New Jersey | Registered: Jul 2005  |  IP: Logged | Report this post to a Moderator
break the chains
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studies have shown that the majority of people with ms tested with pcr come up positive for mycoplasma and lyme easily(1st draw) suggesting an active infection.

http://www.immed.org/illness/autoimmune_illness_research.html#reports
We and others have also found chronic infections in a variety of autoimmune neurologic diseases, such as MS, Lupus (SLE), among others, and neurodegenerative diseases, such as Amyotrophic Lateral Sclerosis (ALS). We previously proposed that many and perhaps a majority of these patients might be suffering from mycoplasmal and other infections that can cause, in part, their complex signs and symptoms. Systemic chronic infections (caused by bacteria such as Mycoplasma, Chlamydia, Borrelia, Brucella, etc. or viruses such as CMV, HHV6, EV or enterovirus, etc.) can dysfunctional immune responses and in extreme cases autoimmune-like disorders. Microorganisms like mycoplasmas can invade virtually every human tissue and can compromise the immune system, permitting opportunistic infections by other bacteria, viruses, fungi and yeast. Mycoplasma, Chlamydia, Borrelia, Rickettsia and other pathogens can also damage and kill nerve cells resulting in nervous system degeneration.

When mycoplasmas exit certain cells, such as synovial cells, nerve cells, among others that can be infected, they can stimulate autoimmune response. This can occur by different mechanisms. One mechanism that has intrigued us is that when certain microorganisms, such as certain species of mycoplasmas, exit from invaded cells, they carry part of the host cell membrane on their surface. This may trigger the immune system to respond to the host antigens on the foreign microorganism. Alternatively, some microorganisms display surface antigens that mimic host cell surface antigens, and these may stimulate autoimmune responses.

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