posted
I can't comment on which is better, but the LLMD that I am seeing prefers for me to use Threelac three times a day. I asked him about theralac and he said that they are completely different. We left it at that.
No yeast issues as of yet. I'm on day 42 of Doxy 400 mg/day.
-------------------- 26 months of treatment. And counting....... Posts: 298 | From Northeast Kansas | Registered: Oct 2006
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lymednva
Frequent Contributor (1K+ posts)
Member # 9098
posted
My LLMD recommends Theralac, and I've found it to be great. When I was unable to get a rx for Diflucan filled due to insurance hassles I doubled up on my Theraclac and that solved the problem.
-------------------- Lymednva Posts: 2407 | From over the river and through the woods | Registered: Apr 2006
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posted
Theralac has been working for me! On recommendation of Lymetoo -I decided to try it-just bought my 3 & 4th bottle-Eight weeks of doxy and so far, so good...and I DO CHEAT on the diet a little -gotta have some carbs .
Posts: 249 | From finger lakes, ny | Registered: Jul 2006
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posted
Theralac seems to be the preferred probiotic.
Nancy do a search on Streptococcus Faecalis (feces) and all the problems it can cause. I dont understand how anyone could take a product with this as an ingredient!!!
Posts: 465 | From New York, NY | Registered: Aug 2005
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posted
I don't feel strongly either way.. but since Streptococcus faecalis was brought up in a negitive way.. I thought I would post some information about it so you don't get incorrect information.
Enterococcus faecalis was formerly classified as Streptococcus faecalis.
The Bacterial Flora of Humans � 2002 Kenneth Todar University of Wisconsin-Madison Department of Bacteriology
The Normal Flora In a healthy animal, the internal tissues, e.g. blood, brain, muscle, etc., are normally free of microorganisms. On the other hand, the surface tissues, e.g. skin and mucous membranes, are constantly in contact with environmental organisms and become readily colonized by certain microbial species. The mixture of organisms regularly found at any anatomical site is referred to as the normal flora.
The normal flora of humans is exceedingly complex and consists of more than 200 species of bacteria. The makeup of the normal flora depends upon various factors, including genetics, age, sex, stress, nutrition and diet of the individual. The normal flora of humans consists of a few eukaryotic fungi and protists, and some methanogenic Archaea that colonize the lower intestinal tract, but the Bacteria are the most numerous and obvious microbial components of the normal flora. The distribution of the bacterial flora of humans is shown in Table 1. This table lists only a fraction of the total bacterial species that occur as normal flora of humans, and it does not express the total number or concentration of bacteria at any site.
Enterococcus faecalis is found in the following, Pharynx, Mouth, Lower Intestine, Anterior urethra and Vagina.
Ask your LLMD on which product they have had success on.
-------------------- 26 months of treatment. And counting....... Posts: 298 | From Northeast Kansas | Registered: Oct 2006
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posted
Vam, not being negative - DNA shows that this once thought harmless bacteria can mutate and cause serious problems. It is not allowed in alternative products in Canada and Australia because of this. Adding 25million CFU every day might be asking for trouble down the road...
Enterococcus faecalis is an opportunistic bacterium that has become one of the most troublesome hospital pathogens. It has intrinsic resistance to many antibiotics and a remarkable capacity for developing resistance to others
Enterococcus faecalis is a Gram-positive, facultatively anaerobic, cocci which occurs singly, in pairs or short chains. It is a normal inhabitant of the intestinal tract and female genital tract.
Scientists have identified a virulence region never seen before in the genome of Enterococcus faecalis - a leading cause of bacterial infection among hospital patients. This bacterium lives peacefully in the human gut, but it also thrives on wounds and burns. Researchers have identified a group of genes that may contribute to the bacterium's transformation from being harmless in the gut to a menacing invader. There has long been concern about the dangers of antibiotic resistance and its implications for the return of infectious diseases that cannot be effectively treated. It is argued that hospitals are not only spreading bacteria from patient to patient, but are also harbouring a tougher breed of bug-one that is resistant to antibiotics.
The sequencing of the genome reinforces knowledge of the remarkable fluidity of the bacterial gene pool. This fluidity allows bacteria to exchange DNA to enhance their ability to cause disease or their resistance to antibiotics.
Generally these microbes live harmlessly in the intestine however until the entire genome was sequenced, scientists did not realise that a quarter of enterococcus' genome is made-up of mobile DNA and within these regions are genes for vancomycin resistance and for virulence. Vancomycin is the last-resort antibiotic in fighting infection.
Enterococcus faecalis is extremely hardy and can survive for weeks on environmental surfaces; cheese - 180 days; soil up to 77 days; soiled linen up to 90 days, cultures at -70? C for several years.
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