-------------------- L.French. Posts: 14 | From France. | Registered: May 2008
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bettyg
Unregistered
posted
french,
i feel if you show BULIMIA in subject by editing your post, you'll get more readers and HELP!
please click on pencil/paper to the side of your 1`st post above,
show on subject line:
BULIMINA, can it cause your weight to increase? ***********************************************
then click EDIT SEND after you went to the LEFT HAND LOWER CORNER and marked for all replies to be sent to you.....
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Hoosiers51
Frequent Contributor (1K+ posts)
Member # 15759
posted
French,
I would say, yes, this is possible. The disease can get in your brain and cause virtually any psychological disorder to come out.
However, are there other symptoms? It would be unusual to have Lyme with bulimia and weight gain only.
It may be hard to be diagnosed with Lyme if those are the only symptoms. Is there a positive blood test?
Posts: 4590 | From Midwest | Registered: Jun 2008
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hshbmom
Frequent Contributor (1K+ posts)
Member # 9478
posted
There is a section in an article by Dr. VTS "Bells Palsy of the Gut" about how Lyme affects the nerves....
It can cause a lack of satiety, or completely the opposite condition.
I've seen both in our family. One person was ready to fight because they were still hungry, even after eating a complete meal. Another time, one person felt like they were full even though they hadn't eaten for hours.
Posts: 1672 | From AL/WV/OH | Registered: Jun 2006
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Keebler
Honored Contributor (25K+ posts)
Member # 12673
posted
-
Bulimia is not listed in the index, still this book may be of some interest:
The Potbelly Syndrome: How Common Germs Cause Obesity, Diabetes, And Heart Disease (Paperback) - 2005
by Russell Farris (Author), Per Marin (Author)
8 customer reviews and you can look inside the book
about $13.00 -
Editorial Reviews
Product Description
Potbelly syndrome (PBS) is a metabolic disorder that affects about one-third of the adults in industrialized countries.
Its most important symptoms are abdominal obesity, high blood pressure, and type 2 diabetes.
Contrary to popular belief,these conditions are caused by chronic infections, not by bad habits. (
PBS is initiated by a small, long-term excess of the stress hormone cortisol.
The extra cortisol stimulates our appetite and slows down our metabolism. It makes fat accumulate in places where it isn't wanted or needed.
Most of the fat settles around our waists, but some of it settles in our liver and muscles.
Liver and muscle cells aren't supposed to store fat, and the fat prevents them from working correctly.
As a result, we feel tired and hungry much of the time. As our potbellies grow and our PBS gets worse, our blood pressure, cholesterol, insulin, and blood sugar levels rise.
Most of the excess cortisol is produced in response to mild, chronic infections. Some of the germs that cause PBS also produce sores in our arteries. When these sores are large enough, they can block arteries and cause heart attacks.
"The Potbelly Syndrome" explains how to diagnose and treat some of the germs that cause PBS and heart disease.
If you've done everything you were supposed to do and still gained weight, became diabetic, or had a heart attack, or if you are a medical professional who suspects that there are serious gaps in the current understanding of obesity, diabetes, and heart disease, "The Potbelly Syndrome" will provide you with the answers you need to bring about better health.
About the Author
Russell Farris is a retired artificial-intelligence researcher who spent most of his life solving problems for the U.S.
Navy. After suffering a heart attack in 1998, he began to apply his problem-solving skills to the study of heart disease and related illnesses.
Per Marin, M.D., Ph.,is a distinguished scientist, physician, and clinical teacher from Sweden. He has been writing about obesity since 1985, and many of his eighty-two publications deal with the effects of cortisol on weight and health.
northstar
Frequent Contributor (1K+ posts)
Member # 7911
posted
(taken from a website for a definition)
quote: What is Bulimia?
Bulimia is characterized by episodes of binge-eating followed by inappropriate methods of weight control (purging).
Inappropriate methods of weight control include vomiting, fasting, enemas, excessive use of laxatives and diuretics, or compulsive exercising.
Excessive shape and weight concerns are also characteristics of bulimia.
A binge is an episode where an individual eats a much larger amount of food than most people would in a similar situation. Binge eating is not a response to intense hunger. It is usually a response to depression, stress, or self esteem issues.
During the binge episode, the individual experiences a loss of control. However, the sense of a loss of control is also followed by a short-lived calmness.
The calmness is often followed by self-loathing. The cycle of overeating and purging usually becomes an obsession and is repeated often.
I could see how periods of ravenous overeating cycling with anorexia, i.e. no signals to eat (not anorexia nervosa) might be interpreted as bulimia.
But bulimia involves self-directed purging or elimination of caloric burden; it does not seem to be just "not feeling hunger".
The other method to compensate for binge eating, exercise, I think might be hard for someone with tick borne diseases.
OCD behavior though, occasionally has been mentioned as a potential manifestation of neuroborelliosis. However, these papers do not go into detail about what actual behavior categories were found. Are there specific kinds of OCD related to these earlier studies? I could not find specifics on this.
Northstar
Posts: 1331 | From hither and yonder | Registered: Sep 2005
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northstar
Frequent Contributor (1K+ posts)
Member # 7911
posted
I did find some studies of bulimia associated with neuroendocrine dysfunction, or other syndromes, on pubmed:
1: involves effect of medications for Parkinson's, not to the disease. The meds affect dopamine levels.
Rev Med Suisse. 2008 May 7;4(156):1145-8, 1150. [Impulse control disorders and Parkinson's disease] [Article in French]
Burkhard PR, Catalano-Chiuv� S, Gronchi-Perrin A, Berney A, Vingerhoets FJ, L�scher C.
Service de neurologie, HUG, Facult� de m�decine de Gen�ve, 1211 Gen�ve.
A variety of behavioral disorders occurring abruptly in patients with Parkinson's disease (PD) has been recently published and attracted considerable attention in the press.
Taking the form of pathological gambling, compulsive shopping, addiction to Internet and to other recreational activities, hypersexuality or bulimia, impulse control disorders (ICD)
related to PD are probably more frequent than previously appreciated and may have consequences as spectacular as disastrous for the involved patients.
ICD are currently viewed as particular adverse reactions to antiparkinsonian medications, notably to dopamine agonists, and, accordingly, tend to improve or disappear when PD therapy is appropriately adjusted.
PMID: 18630168 [PubMed - in process]
2. 1: Neuropsychobiology. 2008 Jun 13;57(3):95-115. [Epub ahead of print]Click here to read Links Stress, Hypothalamic-Pituitary-Adrenal Axis and Eating Disorders. Lo Sauro C, Ravaldi C, Cabras PL, Faravelli C, Ricca V.
Psychiatric Unit, Department of Neuropsychiatric Sciences, University of Florence School of Medicine, University of Florence, Florence, Italy.
The etiopathogenesis of eating disorders (ED) is complex and poorly understood. Biological, psychological and environmental factors have all been considered to be involved in the onset and the persistence of these syndromes, often with conflicting results. The recent literature focused on the possible role of hormonal pathways, in particular the hypothalamic-pituitary-adrenal (HPA) axis, as a relevant factor capable of influencing the onset and the course of ED.
Other studies have suggested that the onset of ED is often preceded by severe life events, and that chronic stress is associated with the persistence of these disorders.
As the biological response to stress is the activation of the HPA axis, the available literature considering the relationships between stress, HPA axis functioning and anorexia nervosa, bulimia nervosa and binge eating disorder is reviewed by the present article. Copyright � 2008 S. Karger AG, Basel.
3.1: J Anxiety Disord. 2008 Feb 29. [Epub ahead of print]Click here to read Links
Eating disorders and obsessive-compulsive disorder: A dimensional approach to purported relations.
Wu KD.
311 Psychology-Computer Science Building, Department of Psychology, Northern Illinois University, DeKalb, IL 60115, United States.
The purpose of this research was to investigate the specificity of purported relations between symptoms of eating disorders (ED) and obsessive-compulsive disorder (OCD).
Whereas most research has focused on diagnostic comorbidity or between-groups analyses, this study took a dimensional approach to investigate specific relations among symptoms of anorexia, bulimia, and OCD, as well as panic, depression, and general distress in a student sample (N=465).
Results were that all symptoms showed significant zero-order correlations, including all ED-OCD pairings.
After removing general distress variance, however, none of three OCD scales significantly predicted anorexia; only compulsive washing among OCD scales significantly predicted bulimia.
Hierarchical multiple regression demonstrated that panic and depression out-performed OCD in predicting bulimia symptoms.
Overall, symptoms of ED and OCD did not show unique relations at the level of core dimensions of each construct. A possible link between bulimia and compulsive washing is worth further study.
PMID: 18396006 [PubMed - as supplied by publisher]
4.1: Psychol Neuropsychiatr Vieil. 2008 Mar;6(1):33-41.Click here to read Links
[Frontotemporal dementia: behavioral story of a neurological disease]
Lebert F, Pasquier F.
Centre de la M�moire, H�pital Salengro, CHRU de Lille, France. [email protected]
The very slow progression of behavioral disorders, initially isolated at the onset of frontotemporal dementia, easily results in their neglect, all the more so since the patients are anosognosic.
In absence of cognitive decline, these patients, whatever carrying a neurological disease, are frequently led towards psychiatrists.
Many psychiatric disorders may be evoked: depression, mania, compulsive obsessive disorder, psychopathy, alcoholic addiction, bulimia, schizophrenia, or Diogene syndrome.
However, the diagnosis can often be easily corrected by a detailed clinical analysis. The knowledge of the 3 behavioral symptoms included in the diagnostic criteria can help to recognize frontotemporal dementia, even when imaging and neuropsychological data show mild abnormalities.
In the last few years, various neuropsychological, biological and environmental mechanisms have been proposed to explain the behavioral disorders.
These disorders are very difficult to tolerate by the caregivers because the patients appear to be asocial and show no affect.
A detailed information of the changes related to the disease is important for the caregivers to accept the behavioral changes and to cope with them. http://tinyurl.
However, over time, the occurrence of mutism may lead caregivers to regret the period of behavioral disorders.
PMID: 18364294 [PubMed - indexed for MEDLINE]
==========================
These were from a list of over 3,000 citations on bulimia neurolyme......but they only gave bulimia since neurolyme (and neuroborreliosis and lyme) did not pull up anything.
posted
My daughter's eating disorder and Lyme story:
Turns out she had undiagnosed Lyme for 7 years and is now being treated by an LLMD. She is now 17.
Before the Lyme diagnosis she had lots of seemingly unrelated symptoms for years: joint and muscle aches, fatigue, hair falling out, headaches, depression, vision issues, ADD, ringing in the ear, insomnia, anxiety ... it's a long list! She missed most of 10th grade and some of 11th graded, was hospitalized 3 times including a psych ward for 6 weeks.
She was diagnosed and treated for an "eating disorder not otherwise specified" meaning she restricted what she ate like anoxeria and purged, like bulimia but without the bingeing.
So we treat the eating disorder like any eating disorder and we treat all the other issues as if Lyme is not the cause. This is mostly because the other 12 or so docs she sees in addition to the LLMDs don't think it's Lyme.
We discovered Lyme was the culprit when I was disgnosed with Lyme and started to understand the symptoms. My husband was also diagnosed with Lyme.
She's been on antibiotics for about 7 months and is remarkably better. The eating disorder docs think their treatment worked, the opthamologist thinks her treatment worked, the psychiatrists think their meds and treatment worked... but the real thing that worked in my mind is the antibiotics.
She still has lots of symptoms and pain and fatigue but her condition has improved. She is not sure that Lyme was the cause of the eating disorder. She says she had body image problems long before the ED came along. So perhaps she had a propensity for ED that Lyme exaggerated and aggravated.
Dr. R. in NYC, Dr. D. in Massachusetts and Dr. G. in Rhode Island (all LLMDs) all said there could be a connection between the purging/restricting behavior but it is not understood.
My anecdotal evidence is that there is a connection between all eating disorders and Lyme!
Posts: 79 | From Rhode Island | Registered: Jun 2008
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