I've heard some things about lyme disease and mercury, mainly that lyme bacteria use mercury to live on? or they sequester (not sure if i spelled that right) it. Is that true?
If so, is one suppose to get rid of mercury first, or kill the lyme first?
When my health problems started 4 years ago I actually found high levels of mercury in my body, wasn't sure if there was any significance with lyme.
I ate a lot of canned tuna growing up but wondered if someone who doesn't have lyme would have gotten rid of that mercury instead of having it collect in the body like in my case.
thanks scott
-------------------- IgM were 31kDa +, 39kDa IND, 41kDa +++, 58kDa +, 66kDa ++ and 83-93kDa ++..
IgG were 31kDa ++, 39kDa IND, 41kDa +++, 58kDa ++. Posts: 30 | From California | Registered: Jul 2009
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Lymeorsomething
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posted
I've heard varying opinions on mercury from LLMDs. The well-known doc I've seen said not to worry about it.
Another said to consider removing my fillings. From what I've read, removing fillings may produce slight to no benefit for the patient.
It probably depends on mercury load too. If you have amalgam fillings, you can't run a chelation protocol as it will pull excess mercury from the fillings supposedly...
If you don't have the fillings, maybe you could try chelation.
My levels don't warrant chelation or filling removal which is expensive and unproven...
-------------------- "Whatever can go wrong will go wrong." Posts: 2062 | From CT | Registered: Jul 2008
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posted
The comprehensive lyme doctors, that means lyme doctors that address all issues not just lyme, mostly say that you can't get rid of lyme if you are mercury toxic. Mercury suppresses the immune system too much along with tons of havoc it causes throughout the body. The comprehensive doctors say that if a patient isn't improving or plateaus then they typically check for heavy metal poisoning, address the metals, and then see improvement.
One of the well know llmd's does ignore mercury and he has a number of patients that are not improving and haven't been for years. Those patients that I know of have tested to be mercury toxic. Yet he still doesn't address it, they still don't improve.
Posts: 499 | From Indiana | Registered: Oct 2007
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seekhelp
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posted
Pyorka, can you PM me and tell me who the LLMD is that ignores it? I want to make sure it's not my doc!! Posts: 7545 | From The 5th Dimension - The Twilight Zone | Registered: Mar 2008
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TerryK
Frequent Contributor (5K+ posts)
Member # 8552
posted
Clin Exp Immunol. 2007 Aug 2;
Mercury exposure as a model for deviation of cytokine responses in experimental Lyme arthritis: HgCl(2) treatment decreases T helper cell type 1-like responses and arthritis severity but delays eradication of Borrelia burgdorferi in C3H/HeN mice.
Ekerfelt C, Andersson M, Olausson A, Bergstr�m S, Hultman P.
Division of Clinical Immunology, and Unit of Autoimmunity and Immune Regulation, Department of Molecular and Clinical Medicine, Faculty of Health Sciences, University Hospital, Link�ping, Sweden.
Lyme borreliosis is a complex infection, where some individuals develop so-called 'chronic borreliosis'. The pathogenetic mechanisms are unknown, but the type of immune response is probably important for healing. A strong T helper cell type 1 (Th1)-like response has been suggested as crucial for eradication of Borrelia and for avoiding development of chronic disease.
Many studies aimed at altering the Th1/Th2 balance in Lyme arthritis employed mice deficient in cytokine genes, but the outcome has not been clear-cut, due possibly to the high redundancy of cytokines.
This study aimed at studying the importance of the Th1/Th2 balance in murine Borrelia arthritis by using the Th2-deviating effect of subtoxic doses of inorganic mercury. Ninety-eight C3H/HeN mice were divided into four groups: Borrelia-infected (Bb), Borrelia-infected exposed to HgCl(2) (BbHg), controls exposed to HgCl(2) alone and normal controls. Mice were killed on days 3, 16, 44 and 65 post-Borrelia inoculation.
Arthritis severity was evaluated by histology, spirochaetal load determined by Borrelia culture, IgG2a- and IgE-levels analysed by enzyme-linked immunosorbemt assay (ELISA) and cytokine-secreting cells detected by enzyme-linked immunospot (ELISPOT).
BbHg mice showed less severe histological arthritis, but delayed eradication of spirochaetes compared to Bb mice, associated with increased levels of IgE (Th2-induced) and decreased levels of IgG2a (Th1-induced), consistent with a Th2-deviation. Both the numbers of Th1 and Th2 cytokine-secreting cells were reduced in BbHg mice, possibly explained by the fact that numbers of cytokine-secreting cells do not correlate with cytokine concentration.
In conclusion, this study supports the hypothesis that a Th1-like response is required for optimal eradication of Borrelia.
PMID: 17672870 [PubMed - as supplied by publisher]
Posts: 6286 | From Oregon | Registered: Jan 2006
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Lymeorsomething
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posted
Treat if tests warrant it but you have to lose the fillings to make it meaningful...
Also plenty are not getting well with normal metal loads...it's just one possible factor...
-------------------- "Whatever can go wrong will go wrong." Posts: 2062 | From CT | Registered: Jul 2008
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Thanks for the replies... I guess ill ask my doc about it when I see him. It sounds like I should find out for sure.
Thanks again Scott
-------------------- IgM were 31kDa +, 39kDa IND, 41kDa +++, 58kDa +, 66kDa ++ and 83-93kDa ++..
IgG were 31kDa ++, 39kDa IND, 41kDa +++, 58kDa ++. Posts: 30 | From California | Registered: Jul 2009
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TerryK
Frequent Contributor (5K+ posts)
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posted
Lymeorsomething wrote: Also plenty are not getting well with normal metal loads...it's just one possible factor...
From the study above "This study aimed at studying the importance of the Th1/Th2 balance in murine Borrelia arthritis by using the Th2-deviating effect of subtoxic doses of inorganic mercury."
Posts: 6286 | From Oregon | Registered: Jan 2006
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