I've suspected I've had LD for awhile now-my hubby (he posts on here occasionally, and we share the same screen name) has been battling this illness for a long time, with little progress.
I had a WB test done last year, and the only band that came up was 41 IGM. I dismissed it, as I had no symptoms, and never worried about it. I had another WB blood test done recently, just for the heck of it, and now the only band present is 41 IGG (chronic late stage LD?)this scares me, making me think I had LD all the time, and it is now in the late stages.
Could this mean that I definitely have LD-or that I was exposed to it, and have no active infection? I can't say I'm symptomless-it's just that my minor symptoms are not debilitating at all, and I never think about them.
Or at least I never thought anything about my possibly having LD Until the emergence of H1N1, my next question:
I've read that anyone with an underlying bacterial infection (like LD?) can get deathly sick from this flu. I usually get a seasonal flu shot every year, but I'm having trouble finding it this year-my Doctor's office is out of the serum, and I'm on a waiting list.
I'm also on the same waiting list for a Pneumonia shot-I have mild asthma, and thought it would be wise to get this vaccine as well. If anyone knows if I could be at high risk for severe complications if I should get the flu with untreated LD, kindly respond as soon as you can. I'm very worried about this!
I have a child in grade K, and my hubby teaches at an elementary school, so the chances of them bringing home the flu, and spreading it to me, are pretty high. At this point there's not much I can do, except hope for the best-
Thanks for reading my long post.
Posts: 64 | From rock tavern ,new york | Registered: Aug 2008
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"With most infections, your immune system first forms IgM antibodies, then in about 2 to 4 weeks, you see IgG antibodies. In some infections, IgG antibodies may be detectable for years.
Because Borrelia burgdorferi is a chronic persistent infection that may last for decades, you would think patients with chronic symptoms would have positive IgG Western blots.
But actually, more IgM blots are positive in chronic borreliosis than IgG. Every time Borrelia burgdorferi reproduces itself, it may stimulate the immune system to form new IgM antibodies.
Some patients have both IgG and IgM blots positive. But if either the IgG or IgM blot is positive, overall it is a positive result."
So .. as you see .. the IgM is more often the indicator that you have chronic Lyme. But either one is important.
-------------------- --Lymetutu-- Opinions, not medical advice! Posts: 96239 | From Texas | Registered: Feb 2001
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posted
Sorry.. I forgot to bring this over on the first reply:
VITAL INFORMATION ON BAND 41!!!!
From a seasoned poster:
"The number of people exposed is in the millions. The reason you can't get treated is they have decided that only early disease merits diagnosis and treatment. The bacterium itself is the most bizarre human bacterial pathogen known, and is poorly understood.
They don't know how many people are carrying a permanent relapsing brain infection. you can't get diagnosis or treatment because they have to pretend it doesn't exist and use labels like 'post lyme' and 'CFS' etc. for those who manifest illness.
Look at what Steere did in his 1992 study which is the foundation for the CDC serodiagnostic standard. He and others often look back on this and refer to a 'normal' control, but in fact the control was taken from sick people...MS sufferers, CFS sufferers, in sum, conditions which could have been caused or complicated by late Bb infection. Moreover, he threw in 25 syphilitic patients which constituted 20% of the control. Hoever, syphilis itself has an annual US incidence of 3 per 100,000.
This statistical chicanery, which fudged the result at 41 kDa on Bb blot by many multiples, is significant, because syph serum will cross react at 41 kDa to Bb western blots. It allowed them to 'swift boat' the importance of the reaction to 41 kDa, which is the earliest and most consistent human ab response to Bb infection, being present in all stages as opposed to the rest of the proteins which are variably expressed according to stage, tissue type, even temperature.
Flagellin {41kDa} is necessary for Bb to survive under all conditions, and is constantly expressed, including in late CNS infection. Yet they chose to swift-boat this response.
Why? It's for political and economic reasons. telling the truth about diagnosis and treatment results in mass panic and probable economic collapse/political revolution. it's likely a bioweapon. North American disease is different from European disease...lack of CSF antibodies, for instance.
A much larger range of serum resistance to host species in wild{allows Bb to infect a much wider range of species, important in disease spread and maintenance in wild}. The CDC has found that Bb 31 goes intracellular in CNS cells.
Telling the truth threatens the careers and livelihoods of the very individuals who control this issue and who have actively lied and deceived and otherwise operated a scientific propaganda campaign for the past 15+ years, profitting from the campaign as they went.
Lyme disease, which in the US also perhaps includes other pathogens notably a bioweaponized bartonella, threatens the entire establishment. If late disease was rare, we'd be able to get treatment. Unfortunately, the EIS/CDC,DOD totally screwed this up and tried to make money off of the disease, making profitability their first priority as opposed to protecting the health of americans.
Think about this...Allen Steere wouldn't listen to Polly Murray in early 90's when she reported a big incidence of neuropsychiatric disease in lyme. She had to call Fallon. Now, Fallon has overwhelming evidence of a serious disabling relapsing brain condition which is not easily treated.
Global hypoperfusion on spect/pet ain't normal folks. Don't you think the CDC etc. should be breaking their balls trying to figure it out? Instead, we see nothing at all, only continued attempts to deny illness and obstruct treatment.
Obviously, they know what is going on, and have determined that the best course is to do nothing, to cover up, knowing that in doing so, they are condemning large numbers of people to perpetual diagnostic and treatment hell.
Think about it. It's a horrific scandal and I'm not sure how much longer these *******s can keep control of it."
-------------------- --Lymetutu-- Opinions, not medical advice! Posts: 96239 | From Texas | Registered: Feb 2001
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I'm going to do an Igenex blood test next-see what that shows, and take it from there!
Posts: 64 | From rock tavern ,new york | Registered: Aug 2008
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-------------------- --Lymetutu-- Opinions, not medical advice! Posts: 96239 | From Texas | Registered: Feb 2001
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Pinelady
Frequent Contributor (5K+ posts)
Member # 18524
posted
donald you may get a better response if you do a trial first. Just a thought.
-------------------- Suspected Lyme 07 Test neg One band migrating in IgG region unable to identify.Igenex Jan.09IFA titer 1:40 IND IgM neg pos 31 +++ 34 IND 39 IND 41 IND 83-93 + DX:Neuroborreliosis Posts: 5850 | From Kentucky | Registered: Dec 2008
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-------------------- --Lymetutu-- Opinions, not medical advice! Posts: 96239 | From Texas | Registered: Feb 2001
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Pinelady
Frequent Contributor (5K+ posts)
Member # 18524
posted
A trial of antibiotics/challenge.
Some have found that when the immune system is shut off by borrelia it will not get a response on the
WB unless there is enough antibodies being made to show positive. When you kill some of them the
body can recognize they are there/attack=antibodies. Some docs now recognize
that some of the worst patients do not make enough antibodies. Because the test is out of
pocket I did this to get more bang for my buck. I was almost in a wheelchair. But yes it is a clinical diagnosis.
-------------------- Suspected Lyme 07 Test neg One band migrating in IgG region unable to identify.Igenex Jan.09IFA titer 1:40 IND IgM neg pos 31 +++ 34 IND 39 IND 41 IND 83-93 + DX:Neuroborreliosis Posts: 5850 | From Kentucky | Registered: Dec 2008
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